Benjamin T. Goetz
Impact in
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- Acute Myeloid Leukemia Research
- Chronic Myeloid Leukemia Treatments
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- Myeloproliferative Neoplasms: Diagnosis and Treatment
Papers in
- Oncology 8
- Cancer Cells and Metastasis 3
- Pancreatic and Hepatic Oncology Research 2
- Peptidase Inhibition and Analysis 1
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- Histone Deacetylase Inhibitors Research 3
- Epigenetics and DNA Methylation 3
- Ubiquitin and proteasome pathways 2
- Co-authors
- Hamid Band (8 shared papers)Wei An (7 shared papers)Bhopal Mohapatra (6 shared papers)Vimla Band (5 shared papers)Neha Zutshi (5 shared papers)Priyanka Arya (4 shared papers)Lin Dong (1 shared paper)Dan Feng (1 shared paper)
- Journals
- iScience (1 paper)Genes & Development (1 paper)The Journal of Immunology (1 paper)Cancer Biology & Therapy (1 paper)Oncotarget (1 paper)
- Partner nations
- United StatesAustralia
In The Last Decade
Benjamin T. Goetz
12 papers receiving 298 citations
Peers
Comparison fields: 5 of 56
- Hematology 61
- Genetics 45
- Immunology 60
- Molecular Biology 184
- Oncology 73
Countries citing papers authored by Benjamin T. Goetz
This map shows the geographic impact of Benjamin T. Goetz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin T. Goetz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin T. Goetz more than expected).
Fields of papers citing papers by Benjamin T. Goetz
This network shows the impact of papers produced by Benjamin T. Goetz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin T. Goetz. The network helps show where Benjamin T. Goetz may publish in the future.
Co-authors
The 25 scholars most cited alongside Benjamin T. Goetz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2012 | 171 | |
| 2 | 2017 | 46 | |
| 3 | 2017 | 16 | |
| 4 | 2020 | 13 | |
| 5 | 2017 | 13 | |
| 6 | 2022 | 11 | |
| 7 | 2020 | 9 | |
| 8 | 2016 | 9 | |
| 9 | 2019 | 9 | |
| 10 | 2024 | 1 | |
| 11 | 2022 | 1 | |
| 12 | 2017 | 1 |
About Benjamin T. Goetz
Benjamin T. Goetz is a scholar working on Oncology, Molecular Biology, Immunology, Genetics and Hematology, having authored 12 papers that have together received 300 indexed citations. Recurring topics across this work include Histone Deacetylase Inhibitors Research (3 papers), Immunotherapy and Immune Responses (3 papers), Cancer Cells and Metastasis (3 papers), Epigenetics and DNA Methylation (3 papers), Pancreatic and Hepatic Oncology Research (2 papers), Ubiquitin and proteasome pathways (2 papers), Peptidase Inhibition and Analysis (1 paper) and Lymphoma Diagnosis and Treatment (1 paper). The work is most often cited by research in Hematology (61 citations), Genetics (45 citations), Immunology (60 citations), Molecular Biology (184 citations) and Oncology (73 citations). Benjamin T. Goetz has collaborated with scholars based in United States and Australia. Frequent co-authors include Hamid Band, Wei An, Bhopal Mohapatra, Vimla Band, Neha Zutshi, Priyanka Arya, Lin Dong, Dan Feng, Nicholas Y. Palermo and Mayumi Naramura. Their work appears in journals such as iScience, Genes & Development, The Journal of Immunology, Cancer Biology & Therapy and Oncotarget.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.