Benjamin Goldenson

1.4k citations
15 papers · 931 · h-index 11

Impact in

Papers in

    • Myeloproliferative Neoplasms: Diagnosis and Treatment 7
    • Chronic Lymphocytic Leukemia Research 3
    • Chronic Myeloid Leukemia Treatments 6
    • Acute Myeloid Leukemia Research 5

Benjamin Goldenson

15 papers receiving 923 citations

Peers

Benjamin Goldenson
Comparison fields: 5 of 81
  • Hematology 232
  • Biological Psychiatry 50
  • Genetics 199
  • Neurology 80
  • Cell Biology 146
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Marie Sanson United States
Rajalakshmi Veeranan‐Karmegam United States
Mikhail A. Kolpakov United States
Jonathan Vanhoutte France
Audrey Noguchi United States
Angela Siciliano Italy
Zhongyuan Bao China
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Citations per field
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Citations per year

Countries citing papers authored by Benjamin Goldenson

Since Specialization
Citations

This map shows the geographic impact of Benjamin Goldenson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Goldenson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Goldenson more than expected).

Fields of papers citing papers by Benjamin Goldenson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Goldenson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Goldenson. The network helps show where Benjamin Goldenson may publish in the future.

Co-authors

The 25 scholars most cited alongside Benjamin Goldenson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Benjamin Goldenson Line = papers co-authored together Benjamin Goldenson links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 2014255
2 2009176
3 2015115
4 200993
5 201363
6 201061
7 200945
8 201537
9 201335
10 201524
11 201119
12 20133
13 20102
14 20112
15 20171

About Benjamin Goldenson

Benjamin Goldenson is a scholar working on Genetics, Hematology, Molecular Biology, Physiology and Biological Psychiatry, having authored 15 papers that have together received 931 indexed citations. Recurring topics across this work include Myeloproliferative Neoplasms: Diagnosis and Treatment (7 papers), Chronic Myeloid Leukemia Treatments (6 papers), Acute Myeloid Leukemia Research (5 papers), Alzheimer's disease research and treatments (3 papers), Chronic Lymphocytic Leukemia Research (3 papers), Microtubule and mitosis dynamics (2 papers), Tryptophan and brain disorders (2 papers) and Neuroinflammation and Neurodegeneration Mechanisms (2 papers). The work is most often cited by research in Hematology (232 citations), Biological Psychiatry (50 citations), Genetics (199 citations), Neurology (80 citations) and Cell Biology (146 citations). Benjamin Goldenson has collaborated with scholars based in United States, Philippines and China. Frequent co-authors include John D. Crispino, Michelle Mahanian, Milan Fiala, Araceli Espinosa‐Jeffrey, Philip T. Liu, John R. Cashman, Xueying Zheng, Qiang Wen, Ken Abel and Senait Ghirmai. Their work appears in journals such as Blood, Nature Medicine, Journal of Alzheimer s Disease, Oncogene and Journal of Neuroimmunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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