Barbara C. Böck

1.9k citations
19 papers · 1.0k · h-index 13

Impact in

Papers in

    • Cell death mechanisms and regulation 5
    • Melanoma and MAPK Pathways 3
    • Protein Kinase Regulation and GTPase Signaling 2
    • Microtubule and mitosis dynamics 2

Barbara C. Böck

19 papers receiving 1000 citations

Peers

Barbara C. Böck
Comparison fields: 5 of 91
  • Oncology 341
  • Cancer Research 166
  • Molecular Biology 645
  • Cell Biology 135
  • Immunology and Allergy 41
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Citations per field
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Citations per year

Countries citing papers authored by Barbara C. Böck

Since Specialization
Citations

This map shows the geographic impact of Barbara C. Böck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara C. Böck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara C. Böck more than expected).

Fields of papers citing papers by Barbara C. Böck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Barbara C. Böck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara C. Böck. The network helps show where Barbara C. Böck may publish in the future.

Co-authors

The 25 scholars most cited alongside Barbara C. Böck, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Barbara C. Böck Line = papers co-authored together Barbara C. Böck links everyone, so they are left out of the graph.

All Works

19 of 19 papers shown
#Work
1 2008179
2 2013165
3 1990160
4 200070
5 200766
6 200665
7 201064
8 200560
9 200751
10 199139
11 201130
12 201425
13 201021
14 20198
15 20166
16 20224
17 20022
18
[The PEA-15 protein induces resistance against glucose deprivation-induced cell death via the ERK/MAP kinase pathway].
20071
19
[Monoclonal antibodies--new probes for diagnosis and therapy. Their use as an example of the micrometastasizing of solid tumors].
19881

About Barbara C. Böck

Barbara C. Böck is a scholar working on Molecular Biology, Cell Biology, Immunology, Oncology and Epidemiology, having authored 19 papers that have together received 1.0k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (5 papers), Phagocytosis and Immune Regulation (3 papers), Melanoma and MAPK Pathways (3 papers), Cell Adhesion Molecules Research (2 papers), Microtubule and mitosis dynamics (2 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Cancer Mechanisms and Therapy (2 papers) and Autophagy in Disease and Therapy (2 papers). The work is most often cited by research in Oncology (341 citations), Cancer Research (166 citations), Molecular Biology (645 citations), Cell Biology (135 citations) and Immunology and Allergy (41 citations). Barbara C. Böck has collaborated with scholars based in Germany, United States and Canada. Frequent co-authors include Wilfried Roth, Kathleen A. Gallo, Jens Witte, Günter Schlimok, G. Riethmüller, I. Funke, Christel Herold‐Mende, Katrin E. Tagscherer, Stephan Macher‐Goeppinger and O. D. Wiestler. Their work appears in journals such as Journal of Biological Chemistry, Molecular Cancer Research, Oncogene, Journal of Clinical Oncology and The Pediatric Infectious Disease Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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