Attila Simor
Impact in
- Biological Psychiatry top 5%
- Tryptophan and brain disorders
- Behavioral Neuroscience top 10%
- Stress Responses and Cortisol
Papers in
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- Mitochondrial Function and Pathology 3
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- Neuroscience and Neuropharmacology Research 3
- Co-authors
- Katalin A. Kékesi (7 shared papers)Gábor Juhász (7 shared papers)Péter Gulyássy (6 shared papers)Éva M. Szegő (2 shared papers)Zoltán Szabó (4 shared papers)Tamás Janáky (4 shared papers)Katalin Völgyi (5 shared papers)Árpád Dobolyi (3 shared papers)
- Journals
- Molecular Neurobiology (2 papers)Journal of Proteomics (1 paper)Molecular and Cellular Neuroscience (1 paper)Brain Behavior and Immunity (1 paper)European Neuropsychopharmacology (1 paper)
- Partner nations
- HungaryAustriaUnited States
In The Last Decade
Attila Simor
7 papers receiving 242 citations
Peers
Comparison fields: 5 of 54
- Biological Psychiatry 66
- Behavioral Neuroscience 31
- Cellular and Molecular Neuroscience 69
- Neurology 30
- Physiology 60
Countries citing papers authored by Attila Simor
This map shows the geographic impact of Attila Simor's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Attila Simor with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Attila Simor more than expected).
Fields of papers citing papers by Attila Simor
This network shows the impact of papers produced by Attila Simor. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Attila Simor. The network helps show where Attila Simor may publish in the future.
Co-authors
The 25 scholars most cited alongside Attila Simor, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 67 | |
| 2 | 2015 | 58 | |
| 3 | 2012 | 57 | |
| 4 | 2013 | 21 | |
| 5 | 2022 | 16 | |
| 6 | 2016 | 16 | |
| 7 | 2017 | 10 |
About Attila Simor
Attila Simor is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Biological Psychiatry, Physiology and Cognitive Neuroscience, having authored 7 papers that have together received 245 indexed citations. Recurring topics across this work include Tryptophan and brain disorders (3 papers), Mitochondrial Function and Pathology (3 papers), Neuroscience and Neuropharmacology Research (3 papers), Sleep and Wakefulness Research (2 papers), Genetics and Neurodevelopmental Disorders (1 paper), Circadian rhythm and melatonin (1 paper), Alzheimer's disease research and treatments (1 paper) and Biochemical effects in animals (1 paper). The work is most often cited by research in Biological Psychiatry (66 citations), Behavioral Neuroscience (31 citations), Cellular and Molecular Neuroscience (69 citations), Neurology (30 citations) and Physiology (60 citations). Attila Simor has collaborated with scholars based in Hungary, Austria and United States. Frequent co-authors include Katalin A. Kékesi, Gábor Juhász, Péter Gulyássy, Éva M. Szegő, Zoltán Szabó, Tamás Janáky, Katalin Völgyi, Árpád Dobolyi, Géza Müller and András Czurkó. Their work appears in journals such as Molecular Neurobiology, Journal of Proteomics, Molecular and Cellular Neuroscience, Brain Behavior and Immunity and European Neuropsychopharmacology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.