Åsa Thulin

480 citations
12 papers · 380 · h-index 11

Impact in

Papers in

    • Extracellular vesicles in disease 4
    • Angiogenesis and VEGF in Cancer 2
    • Glycosylation and Glycoproteins Research 1
    • Inflammatory Biomarkers in Disease Prognosis 4

Åsa Thulin

12 papers receiving 369 citations

Peers

Åsa Thulin
Comparison fields: 5 of 63
  • Immunology and Allergy 31
  • Internal Medicine 17
  • Cancer Research 73
  • Hematology 51
  • Immunology 78
Replace Goedele Wallays with:
Goedele Wallays Belgium
Benedikt Weitkamp Germany
Ghislaine Chérel France
Christine Edmead United Kingdom
Ann Pascale Bijnens Netherlands
Valerie J. Ludbrook United Kingdom
Alireza Ebrahimnejad Germany
J.‐H. SHIEH United States
Alexander Martens Germany
Åsa Thulin relative to Goedele Wallays Belgium Goedele Wallays's profile →
Citations per field
00.5×12.3×
Goedele Wallays · 1×
Citations per year

Countries citing papers authored by Åsa Thulin

Since Specialization
Citations

This map shows the geographic impact of Åsa Thulin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Åsa Thulin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Åsa Thulin more than expected).

Fields of papers citing papers by Åsa Thulin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Åsa Thulin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Åsa Thulin. The network helps show where Åsa Thulin may publish in the future.

Co-authors

The 25 scholars most cited alongside Åsa Thulin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Åsa Thulin Line = papers co-authored together Åsa Thulin links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 202066
2 200655
3 201744
4 201741
5 200939
6 200634
7 201627
8 201322
9 201119
10 202016
11 201611
12 20186

About Åsa Thulin

Åsa Thulin is a scholar working on Molecular Biology, Oncology, Cancer Research, Immunology and Allergy and Cardiology and Cardiovascular Medicine, having authored 12 papers that have together received 380 indexed citations. Recurring topics across this work include Inflammatory Biomarkers in Disease Prognosis (4 papers), Extracellular vesicles in disease (4 papers), Cell Adhesion Molecules Research (3 papers), Angiogenesis and VEGF in Cancer (2 papers), Protease and Inhibitor Mechanisms (2 papers), Cardiovascular Disease and Adiposity (2 papers), Prostate Cancer Treatment and Research (1 paper) and Glycosylation and Glycoproteins Research (1 paper). The work is most often cited by research in Immunology and Allergy (31 citations), Internal Medicine (17 citations), Cancer Research (73 citations), Hematology (51 citations) and Immunology (78 citations). Åsa Thulin has collaborated with scholars based in Sweden, Finland and Germany. Frequent co-authors include Agneta Siegbahn, Anna‐Karin Olsson, Christina Christersson, Lena Claesson‐Welsh, Chunsik Lee, Johan Dixelius, Agneta Siegbahn, Irja Johansson, Jessica Cedervall and Maria Ringvall. Their work appears in journals such as Thrombosis and Haemostasis, Cancer Research, Biomarkers in Medicine, Angiogenesis and Thrombosis Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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