Alfred Lanzafame

551 citations
15 papers · 462 · h-index 9

Impact in

Papers in

    • Receptor Mechanisms and Signaling 8
    • Ion channel regulation and function 6
    • Protein Kinase Regulation and GTPase Signaling 2
    • Ion Transport and Channel Regulation 1
    • Phosphodiesterase function and regulation 1
    • Neuroscience and Neuropharmacology Research 4

Alfred Lanzafame

13 papers receiving 442 citations

Peers

Alfred Lanzafame
Comparison fields: 5 of 65
  • Cellular and Molecular Neuroscience 241
  • Aging 9
  • Molecular Biology 345
  • Physiology 23
  • Endocrine and Autonomic Systems 17
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Citations per year

Countries citing papers authored by Alfred Lanzafame

Since Specialization
Citations

This map shows the geographic impact of Alfred Lanzafame's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alfred Lanzafame with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alfred Lanzafame more than expected).

Fields of papers citing papers by Alfred Lanzafame

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alfred Lanzafame. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alfred Lanzafame. The network helps show where Alfred Lanzafame may publish in the future.

Co-authors

The 22 scholars most cited alongside Alfred Lanzafame, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Alfred Lanzafame Line = papers co-authored together Alfred Lanzafame links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1 2003111
2 199899
3 200374
4 200445
5 200626
6 199626
7 199723
8 200521
9 200420
10 20117
11 20017
12 20122
13 19991
14 20250
15 20210

About Alfred Lanzafame

Alfred Lanzafame is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Surgery, Pharmacology and Pathology and Forensic Medicine, having authored 15 papers that have together received 462 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (8 papers), Ion channel regulation and function (6 papers), Neuroscience and Neuropharmacology Research (4 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Cardiac electrophysiology and arrhythmias (1 paper), Circadian rhythm and melatonin (1 paper), Ion Transport and Channel Regulation (1 paper) and Phosphodiesterase function and regulation (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (241 citations), Aging (9 citations), Molecular Biology (345 citations), Physiology (23 citations) and Endocrine and Autonomic Systems (17 citations). Alfred Lanzafame has collaborated with scholars based in Australia, Japan and United States. Frequent co-authors include Arthur Christopoulos, F. Mitchelson, Patrick M. Sexton, Elizabeth Guida, Albrecht Ziegler, Jane F. Arthur, Lynne Turnbull, Elizabeth A. Woodcock, Catherine Kirby and Susan R. Davis. Their work appears in journals such as Biochemical Pharmacology, European Journal of Pharmacology, Life Sciences, Journal of Pharmacology and Experimental Therapeutics and Molecular Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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