A. Massa

713 citations
41 papers · 538 · h-index 12

Impact in

  • Genetics top 5%
    • Hemoglobinopathies and Related Disorders
    • Myeloproliferative Neoplasms: Diagnosis and Treatment

Papers in

    • Hemoglobinopathies and Related Disorders 20
    • Iron Metabolism and Disorders 7
    • Blood groups and transfusion 5

A. Massa

33 papers receiving 506 citations

Peers

A. Massa
Comparison fields: 5 of 92
  • Genetics 206
  • Family Practice 20
  • Hematology 121
  • Cell Biology 119
  • Infectious Diseases 80
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Citations per field
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Citations per year

Countries citing papers authored by A. Massa

Since Specialization
Citations

This map shows the geographic impact of A. Massa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Massa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Massa more than expected).

Fields of papers citing papers by A. Massa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Massa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Massa. The network helps show where A. Massa may publish in the future.

Co-authors

The 25 scholars most cited alongside A. Massa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. Massa Line = papers co-authored together A. Massa links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 41 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2006129
2 197382
3 201042
4 200038
5 198931
6 198425
7 199622
8 197120
9 197917
10 197615
11
Respiratory dysfunction related to diaphragmatic shoulder pain after abdominal and pelvic laparoscopy.
199613
12 197912
13 198111
14 198311
15 19849
16 20198
17
Behaviour of several 'progression markers' during the HIV-Ab seroconversion period. Comparison with later stages.
19928
18 20227
19 19837
20 19816

About A. Massa

A. Massa is a scholar working on Genetics, Hematology, Cell Biology, Communication and Molecular Biology, having authored 41 papers that have together received 538 indexed citations. Recurring topics across this work include Hemoglobinopathies and Related Disorders (20 papers), Iron Metabolism and Disorders (7 papers), Hemoglobin structure and function (7 papers), Blood groups and transfusion (5 papers), Social Media and Politics (4 papers), Media Studies and Communication (4 papers), Neonatal Health and Biochemistry (3 papers) and Erythrocyte Function and Pathophysiology (3 papers). The work is most often cited by research in Genetics (206 citations), Family Practice (20 citations), Hematology (121 citations), Cell Biology (119 citations) and Infectious Diseases (80 citations). A. Massa has collaborated with scholars based in Italy, United States and Bulgaria. Frequent co-authors include K. Rivet Amico, Fabrizio Starace, Jeffrey D. Fisher, L Tentori, M Marinucci, Eraldo Antonini, S. Carta, Maurizio Brunori, Fulvio Mavilio and Sergio Amadori. Their work appears in journals such as British Journal of Haematology, Blood, Journal of Insect Physiology, Acta Haematologica and Human Heredity.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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