A. E. Irvine

513 citations
9 papers · 417 · h-index 8

Impact in

    • Neutrophil, Myeloperoxidase and Oxidative Mechanisms
    • Immune Response and Inflammation
    • T-cell and B-cell Immunology
    • Immune Cell Function and Interaction
    • Phagocytosis and Immune Regulation
  • Rheumatology top 10%
    • Systemic Lupus Erythematosus Research

Papers in

A. E. Irvine

9 papers receiving 408 citations

Peers

A. E. Irvine
Comparison fields: 5 of 64
  • Immunology 202
  • Rheumatology 116
  • Hematology 83
  • Immunology and Allergy 26
  • Oncology 82
Replace Belinda Baltus with:
Belinda Baltus Netherlands
Do‐June Min South Korea
L. A. M. Van Den Bersselaar Netherlands
T. Saber Ireland
JR Kalden Germany
Holger Ruchatz United Kingdom
Hitoshi Deguchi Japan
Naoko Yoshio‐Hoshino Japan
Renate E. Gay United States
J R Kalden Germany
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Citations per field
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Citations per year

Countries citing papers authored by A. E. Irvine

Since Specialization
Citations

This map shows the geographic impact of A. E. Irvine's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. E. Irvine with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. E. Irvine more than expected).

Fields of papers citing papers by A. E. Irvine

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. E. Irvine. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. E. Irvine. The network helps show where A. E. Irvine may publish in the future.

Co-authors

The 23 scholars most cited alongside A. E. Irvine, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. E. Irvine Line = papers co-authored together A. E. Irvine links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 1999183
2 199765
3 199541
4 201034
5 200831
6 199925
7 199523
8
Human umbilical cord conditioned medium: a stimulus for human CFU-G.
198412
9 20083

About A. E. Irvine

A. E. Irvine is a scholar working on Immunology, Surgery, Molecular Biology, Oncology and Immunology and Allergy, having authored 9 papers that have together received 417 indexed citations. Recurring topics across this work include Cell Adhesion Molecules Research (2 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (1 paper), Cancer Genomics and Diagnostics (1 paper), Protein Degradation and Inhibitors (1 paper), Bone health and treatments (1 paper), Musculoskeletal pain and rehabilitation (1 paper), Mesenchymal stem cell research (1 paper) and Rheumatoid Arthritis Research and Therapies (1 paper). The work is most often cited by research in Immunology (202 citations), Rheumatology (116 citations), Hematology (83 citations), Immunology and Allergy (26 citations) and Oncology (82 citations). A. E. Irvine has collaborated with scholars based in United Kingdom. Frequent co-authors include A. L. Bell, K Williamson, A. D. Crockard, P A Courtney, Rebekah Kennedy, T. C. M. Morris, W R McKane, Mary Drake, Z. R. Desai and L. Nolan. Their work appears in journals such as Annals of the Rheumatic Diseases, Blood Reviews, British Journal of Haematology, European Journal of Orthopaedic Surgery & Traumatology and Leukemia Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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