Hitoshi Deguchi
Impact in
- Rheumatology top 10%
- Rheumatoid Arthritis Research and Therapies
- Systemic Lupus Erythematosus Research
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- Immune Cell Function and Interaction
- T-cell and B-cell Immunology
- Immunodeficiency and Autoimmune Disorders
Papers in
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- T-cell and B-cell Immunology 4
- Immune Cell Function and Interaction 4
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- Monoclonal and Polyclonal Antibodies Research 4
- Co-authors
- Tadamitsu Kishimoto (5 shared papers)Masaki Suemura (4 shared papers)Keiji Maeda (1 shared paper)Norihiro Nishimoto (1 shared paper)Nobuhiro Takagi (1 shared paper)Nobuyuki Imai (1 shared paper)Kazuyuki Yoshizaki (1 shared paper)Taro Kuritani (1 shared paper)
In The Last Decade
Hitoshi Deguchi
5 papers receiving 315 citations
Peers
Comparison fields: 5 of 62
- Rheumatology 130
- Immunology 140
- Hematology 49
- Radiology, Nuclear Medicine and Imaging 72
- Immunology and Allergy 15
Countries citing papers authored by Hitoshi Deguchi
This map shows the geographic impact of Hitoshi Deguchi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hitoshi Deguchi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hitoshi Deguchi more than expected).
Fields of papers citing papers by Hitoshi Deguchi
This network shows the impact of papers produced by Hitoshi Deguchi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hitoshi Deguchi. The network helps show where Hitoshi Deguchi may publish in the future.
Co-authors
The 25 scholars most cited alongside Hitoshi Deguchi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Toxicity, pharmacokinetics, and dose-finding study of repetitive treatment with the humanized anti-interleukin 6 receptor antibody MRA in rheumatoid arthritis. Phase I/II clinical study. | 2003 | 166 |
| 2 | 2005 | 97 | |
| 3 | 1983 | 47 | |
| 4 | 1981 | 32 | |
| 5 | Regulation of human IgE response by IgE class-specific suppressor T cells or suppressor factor(s). | 1983 | 1 |
| 6 | 1982 | 0 |
About Hitoshi Deguchi
Hitoshi Deguchi is a scholar working on Immunology, Radiology, Nuclear Medicine and Imaging, Rheumatology, Infectious Diseases and Organic Chemistry, having authored 6 papers that have together received 343 indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (4 papers), Immune Cell Function and Interaction (4 papers), Monoclonal and Polyclonal Antibodies Research (4 papers), Rheumatoid Arthritis Research and Therapies (1 paper) and Systemic Lupus Erythematosus Research (1 paper). The work is most often cited by research in Rheumatology (130 citations), Immunology (140 citations), Hematology (49 citations), Radiology, Nuclear Medicine and Imaging (72 citations) and Immunology and Allergy (15 citations). Hitoshi Deguchi has collaborated with scholars based in Japan and France. Frequent co-authors include Tadamitsu Kishimoto, Masaki Suemura, Keiji Maeda, Norihiro Nishimoto, Nobuhiro Takagi, Nobuyuki Imai, Kazuyuki Yoshizaki, Taro Kuritani, Yûichi Yamamura and Akitoshi Ishizaka. Their work appears in journals such as The Journal of Immunology, Current topics in microbiology and immunology, DNA Research and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.