Yuko Itoh

2.5k citations
78 papers · 2.1k · h-index 23

Impact in

Papers in

    • Genomics and Phylogenetic Studies 5
    • RNA and protein synthesis mechanisms 5
    • Microbial Community Ecology and Physiology 5

Yuko Itoh

74 papers receiving 2.1k citations

Peers

Yuko Itoh
Comparison fields: 5 of 131
  • Environmental Chemistry 242
  • Cancer Research 259
  • Molecular Biology 1.2k
  • Ecology 315
  • Cell Biology 119
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Citations per field
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Citations per year

Countries citing papers authored by Yuko Itoh

Since Specialization
Citations

This map shows the geographic impact of Yuko Itoh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yuko Itoh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yuko Itoh more than expected).

Fields of papers citing papers by Yuko Itoh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yuko Itoh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yuko Itoh. The network helps show where Yuko Itoh may publish in the future.

Co-authors

The 25 scholars most cited alongside Yuko Itoh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Yuko Itoh Line = papers co-authored together Yuko Itoh links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 78 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Induction of apoptosis as well as necrosis by hypoxia and predominant prevention of apoptosis by Bcl-2 and Bcl-XL.
1996329
2 2010215
3
Marked prevention of tumor growth and metastasis by a novel immunosuppressive agent, FTY720, in mouse breast cancer models.
2002178
4 2001152
5 199896
6 200592
7 197880
8 198761
9 199556
10 200456
11 200453
12 200044
13 201443
14 197141
15 200038
16 198633
17 200129
18 200028
19 200327
20 199225

About Yuko Itoh

Yuko Itoh is a scholar working on Molecular Biology, Ecology, Plant Science, Global and Planetary Change and Environmental Chemistry, having authored 78 papers that have together received 2.1k indexed citations. Recurring topics across this work include Lipid metabolism and biosynthesis (6 papers), Genomics and Phylogenetic Studies (5 papers), Forest ecology and management (5 papers), RNA and protein synthesis mechanisms (5 papers), Microbial Community Ecology and Physiology (5 papers), Lichen and fungal ecology (4 papers), Carbohydrate Chemistry and Synthesis (4 papers) and Tree-ring climate responses (4 papers). The work is most often cited by research in Environmental Chemistry (242 citations), Cancer Research (259 citations), Molecular Biology (1.2k citations), Ecology (315 citations) and Cell Biology (119 citations). Yuko Itoh has collaborated with scholars based in Japan, United States and Indonesia. Frequent co-authors include Akihiko Sugai, Toshihiro Itoh, Ikuko Uda, Norio Kurosawa, Tomohiro Itoh, Wataru Kamiike, Yawara Eguchi, Satoshi Shimizu, Hikaru Matsuda and Junichi Hasegawa. Their work appears in journals such as Extremophiles, Proceedings of the National Academy of Sciences, Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, Forests and ACS Chemical Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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