William J. DeVito

57 papers receiving 1.0k citations

Peers

William J. DeVito
Comparison fields: 5 of 84
  • Behavioral Neuroscience 127
  • Endocrine and Autonomic Systems 158
  • Endocrinology, Diabetes and Metabolism 372
  • Reproductive Medicine 154
  • Developmental Neuroscience 72
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Citations per year

Countries citing papers authored by William J. DeVito

Since Specialization
Citations

This map shows the geographic impact of William J. DeVito's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William J. DeVito with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William J. DeVito more than expected).

Fields of papers citing papers by William J. DeVito

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William J. DeVito. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William J. DeVito. The network helps show where William J. DeVito may publish in the future.

Co-authors

The 25 scholars most cited alongside William J. DeVito, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with William J. DeVito Line = papers co-authored together William J. DeVito links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 57 papers — load more, or switch the sort, to bring in the rest.

#Work
1 200572
2 199264
3 199260
4 199559
5 199653
6 199345
7 199541
8 200438
9 199535
10 200834
11 199231
12 198530
13 198026
14 199926
15 199124
16
Induction of transcription factor interferon regulatory factor-1 by interferon-gamma (IFN gamma) and tumor necrosis factor-alpha (TNF alpha) in FRTL-5 cells.
199922
17 199721
18 198820
19 199620
20 198920

About William J. DeVito

William J. DeVito is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology, Behavioral Neuroscience, Reproductive Medicine and Oncology, having authored 57 papers that have together received 1.1k indexed citations. Recurring topics across this work include Growth Hormone and Insulin-like Growth Factors (16 papers), Stress Responses and Cortisol (12 papers), Hypothalamic control of reproductive hormones (11 papers), Thyroid Disorders and Treatments (11 papers), Neuroendocrine regulation and behavior (6 papers), Immune Response and Inflammation (6 papers), Neuroinflammation and Neurodegeneration Mechanisms (5 papers) and Birth, Development, and Health (5 papers). The work is most often cited by research in Behavioral Neuroscience (127 citations), Endocrine and Autonomic Systems (158 citations), Endocrinology, Diabetes and Metabolism (372 citations), Reproductive Medicine (154 citations) and Developmental Neuroscience (72 citations). William J. DeVito has collaborated with scholars based in United States, Greece and Sweden. Frequent co-authors include Lewis E. Braverman, S Stone, William C. Okulicz, George A. Hedge, John M. Connors, Kuo‐Tung Tang, Kouki Mori, Kam‐Tsun Tang, James R. Sutterer and C I. Their work appears in journals such as Endocrinology, Journal of Cellular Biochemistry, Neuroendocrinology, Psychosomatic Medicine and Biochemical and Biophysical Research Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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