Weinstein Ib

2.2k citations
51 papers · 2k · h-index 25

Impact in

    • Carcinogens and Genotoxicity Assessment
    • Protease and Inhibitor Mechanisms
  • Oncology top 10%
    • Cancer-related Molecular Pathways

Papers in

Weinstein Ib

51 papers receiving 1.8k citations

Peers

Weinstein Ib
Comparison fields: 5 of 82
  • Cancer Research 476
  • Oncology 353
  • Molecular Biology 954
  • Pathology and Forensic Medicine 141
  • Immunology 114
Replace Ken Brown with:
Ken Brown United Kingdom
Marco Ricci United States
Robert L. Sutherland Australia
Cécile Demur France
S H Yuspa United States
Esther Landesman‐Bollag United States
Isaac Berenblum Israel
Mark E. Stearns United States
Jean M. Gudas United States
David T. Kiang United States
Weinstein Ib relative to Ken Brown United Kingdom Ken Brown's profile →
Citations per field
00.5×1.5×
Ken Brown · 1×
Citations per year

Countries citing papers authored by Weinstein Ib

Since Specialization
Citations

This map shows the geographic impact of Weinstein Ib's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Weinstein Ib with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Weinstein Ib more than expected).

Fields of papers citing papers by Weinstein Ib

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Weinstein Ib. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Weinstein Ib. The network helps show where Weinstein Ib may publish in the future.

Co-authors

The 25 scholars most cited alongside Weinstein Ib, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Weinstein Ib Line = papers co-authored together Weinstein Ib links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 51 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1996280
2
The epsilon isoform of protein kinase C is an oncogene when overexpressed in rat fibroblasts.
1993206
3
Antisense to cyclin D1 inhibits growth and reverses the transformed phenotype of human esophageal cancer cells.
1995134
4
Altered levels of protein kinase C and Ca2+-dependent protein kinases in human colon carcinomas.
1987117
5
Effect of N-2-acetylaminofluorene modification on the conformation of nucleic acids.
197499
6
Differential excision from DNA of the C-8 and N2 guanosine adducts of N-acetyl-2-aminofluorene by single strand-specific endonucleases.
197797
7
Expression of cyclins D1 and E in human colon adenocarcinomas.
199770
8
Linking protein kinase C to the cell cycle: ectopic expression of PKC eta in NIH3T3 cells alters the expression of cyclins and Cdk inhibitors and induces adipogenesis.
199664
9
Ultraviolet light induces the expression of oncogenes in rat fibroblast and human keratinocyte cells.
198863
10
Disturbances in growth control and gene expression in a C3H/10T1/2 cell line that stably overproduces protein kinase C.
198959
11
Immunological quantitation of levels of tissue inhibitor of metalloproteinase-1 in human colon cancer.
199153
12
Binding of [3H]benzo(a)pyrene to natural and synthetic nucleic acids in a subcellular microsomal system.
197552
13
Heterogeneity of murine erythroleukemia cells with respect to tumor promoter-mediated inhibition of cell differentiation.
197848
14
Expression of retroviral sequences and oncogenes in rat liver tumors induced by diethylnitrosamine.
198747
15
Induction of chemotaxis in mouse peritoneal macrophages by phorbol ester tumor promoters.
198145
16
1 alpha,25-dihydroxyvitamin D3-induced regulation of protein kinase C gene expression during HL-60 cell differentiation.
199144
17
Modulation of phagocytosis by tumor promoters and epidermal growth factor in normal and transformed macrophages.
198043
18
Structure-activity studies on the mechanism of action of miracil D.
196837
19
The role of protein kinase C in growth control and the concept of carcinogenesis as a progressive disorder in signal transduction.
199037
20
Effect of N-2-acetylaminofluorene modification on the structure and template activity of DNA and reconstituted chromatin.
197736

About Weinstein Ib

Weinstein Ib is a scholar working on Cancer Research, Oncology, Molecular Biology, Immunology and Pharmacology, having authored 51 papers that have together received 2k indexed citations. Recurring topics across this work include Carcinogens and Genotoxicity Assessment (21 papers), Protein Kinase Regulation and GTPase Signaling (12 papers), DNA and Nucleic Acid Chemistry (5 papers), Cancer-related Molecular Pathways (5 papers), Cancer and biochemical research (4 papers), Inflammatory mediators and NSAID effects (3 papers), T-cell and Retrovirus Studies (3 papers) and Immune cells in cancer (3 papers). The work is most often cited by research in Cancer Research (476 citations), Oncology (353 citations), Molecular Biology (954 citations), Pathology and Forensic Medicine (141 citations) and Immunology (114 citations). Weinstein Ib has collaborated with scholars based in United States, Taiwan and Israel. Frequent co-authors include Dezider Grünberger, Nadir Arber, Doriano Fabbro, Sarah Nichols Guadagno, Peter R. Holt, Silu Wang, Hanina H. Hibshoosh, Melissa Dowd Begg, Ira Schieren and Peter Pulkrabek. Their work appears in journals such as Gastroenterology, British Journal of Cancer, Molecular Pharmacology and PubMed.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact