Walker R. Force

8 papers receiving 1.6k citations

Walker R. Force's Hit Papers

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas 1995 · 1.2k citations
1.2k0+10+20Years since publication2505007501k

Peers

Walker R. Force
Comparison fields: 5 of 61
  • Immunology 579
  • Cancer Research 210
  • Virology 65
  • Molecular Biology 727
  • Oncology 90
Replace David M. Calderhead with:
David M. Calderhead United States
Hui Hu United States
Oliver Kemper Israel
Nigel Sharfe Canada
G L Shu United States
A Ishii Japan
Nicole Avitahl United States
Eva M. Nilsson Sweden
R Remy Switzerland
Beata Majchrzak Canada
Walker R. Force relative to David M. Calderhead United States David M. Calderhead's profile →
Citations per field
00.5×1.5×1.9×
David M. Calderhead · 1×
Citations per year

Countries citing papers authored by Walker R. Force

Since Specialization
Citations

This map shows the geographic impact of Walker R. Force's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Walker R. Force with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Walker R. Force more than expected).

Fields of papers citing papers by Walker R. Force

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Walker R. Force. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Walker R. Force. The network helps show where Walker R. Force may publish in the future.

Co-authors

The 25 scholars most cited alongside Walker R. Force, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Walker R. Force Line = papers co-authored together Walker R. Force links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1
Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas
Hit paper breakdown →
19951195
2 1997153
3 1995112
4 199778
5 200052
6 199240
7 199428
8 199417

About Walker R. Force

Walker R. Force is a scholar working on Cancer Research, Endocrinology, Diabetes and Metabolism, Genetics, Molecular Biology and Immunology, having authored 8 papers that have together received 1.7k indexed citations. Recurring topics across this work include NF-κB Signaling Pathways (4 papers), Thyroid Disorders and Treatments (3 papers), Estrogen and related hormone effects (3 papers), Genomics and Chromatin Dynamics (3 papers), Cell death mechanisms and regulation (2 papers), T-cell and B-cell Immunology (1 paper) and Immune Response and Inflammation (1 paper). The work is most often cited by research in Immunology (579 citations), Cancer Research (210 citations), Virology (65 citations), Molecular Biology (727 citations) and Oncology (90 citations). Walker R. Force has collaborated with scholars based in United States. Frequent co-authors include Carl F. Ware, Artin Mahboubi, Nam Jin Yoo, Douglas R. Green, David Lynch, Thomas Brunner, Rona J. Mogil, Séamus J. Martin, Drake LaFace and Fernando Echeverri. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology, Nature, Proceedings of the National Academy of Sciences and Molecular Endocrinology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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