Ute Nonhoff
Impact in
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- Genetic Neurodegenerative Diseases
- Neurology top 10%
- Amyotrophic Lateral Sclerosis Research
Papers in
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- RNA Research and Splicing 4
- Mitochondrial Function and Pathology 4
- bioluminescence and chemiluminescence research 1
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- Genetic Neurodegenerative Diseases 5
- Co-authors
- Sylvia Krobitsch (6 shared papers)Hans Lehrach (5 shared papers)Markus Ralser (4 shared papers)Franziska Welzel (2 shared papers)Daniela Balzereit (1 shared paper)Marie‐Laure Yaspo (1 shared paper)Ilaria Piccini (1 shared paper)Thomas Lengauer (2 shared papers)
- Journals
- Human Molecular Genetics (2 papers)Molecular Biology of the Cell (1 paper)Arthritis Research & Therapy (1 paper)Annals of the Rheumatic Diseases (1 paper)PLoS ONE (1 paper)
- Partner nations
- GermanySwedenNetherlands
In The Last Decade
Ute Nonhoff
8 papers receiving 647 citations
Peers
Comparison fields: 5 of 62
- Cellular and Molecular Neuroscience 283
- Neurology 181
- Genetics 85
- Molecular Biology 513
- Aging 8
Countries citing papers authored by Ute Nonhoff
This map shows the geographic impact of Ute Nonhoff's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ute Nonhoff with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ute Nonhoff more than expected).
Fields of papers citing papers by Ute Nonhoff
This network shows the impact of papers produced by Ute Nonhoff. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ute Nonhoff. The network helps show where Ute Nonhoff may publish in the future.
Co-authors
The 25 scholars most cited alongside Ute Nonhoff, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 276 | |
| 2 | 2004 | 126 | |
| 3 | 2012 | 84 | |
| 4 | 2005 | 81 | |
| 5 | 2020 | 39 | |
| 6 | 2010 | 33 | |
| 7 | 2021 | 8 | |
| 8 | 2004 | 2 |
About Ute Nonhoff
Ute Nonhoff is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Pharmacology, Cardiology and Cardiovascular Medicine and Oncology, having authored 8 papers that have together received 649 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (5 papers), RNA Research and Splicing (4 papers), Mitochondrial Function and Pathology (4 papers), bioluminescence and chemiluminescence research (1 paper), Cholinesterase and Neurodegenerative Diseases (1 paper), Systemic Lupus Erythematosus Research (1 paper), Computational Drug Discovery Methods (1 paper) and Oral microbiology and periodontitis research (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (283 citations), Neurology (181 citations), Genetics (85 citations), Molecular Biology (513 citations) and Aging (8 citations). Ute Nonhoff has collaborated with scholars based in Germany, Sweden and Netherlands. Frequent co-authors include Sylvia Krobitsch, Hans Lehrach, Markus Ralser, Franziska Welzel, Daniela Balzereit, Marie‐Laure Yaspo, Ilaria Piccini, Thomas Lengauer, Mario Albrecht and Jörg Isensee. Their work appears in journals such as Human Molecular Genetics, Molecular Biology of the Cell, Arthritis Research & Therapy, Annals of the Rheumatic Diseases and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.