Torsten Exner
Impact in
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- Receptor Mechanisms and Signaling
- Protein Kinase Regulation and GTPase Signaling
- PI3K/AKT/mTOR signaling in cancer
- Ion channel regulation and function
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- Neuroscience and Neuropharmacology Research
Papers in
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- Receptor Mechanisms and Signaling 6
- Protein Kinase Regulation and GTPase Signaling 4
- PI3K/AKT/mTOR signaling in cancer 2
- Ion channel regulation and function 1
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- Cellular transport and secretion 1
- Co-authors
- Bernd Nürnberg (9 shared papers)Udo Maier (2 shared papers)Daniela Leopoldt (2 shared papers)Theodor Hanck (1 shared paper)Reinhard Wetzker (1 shared paper)Patricia Viard (1 shared paper)Jean Mironneau (1 shared paper)Nathalie Macrez (1 shared paper)
- Journals
- Biochemical Journal (2 papers)Journal of Medicinal Chemistry (1 paper)European Journal of Biochemistry (1 paper)Analytical Biochemistry (1 paper)The FASEB Journal (1 paper)
- Partner nations
- GermanyFranceSwitzerland
In The Last Decade
Torsten Exner
9 papers receiving 441 citations
Peers
Comparison fields: 5 of 49
- Molecular Biology 320
- Cellular and Molecular Neuroscience 50
- Physiology 12
- Cell Biology 32
- Sensory Systems 12
Countries citing papers authored by Torsten Exner
This map shows the geographic impact of Torsten Exner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Torsten Exner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Torsten Exner more than expected).
Fields of papers citing papers by Torsten Exner
This network shows the impact of papers produced by Torsten Exner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Torsten Exner. The network helps show where Torsten Exner may publish in the future.
Co-authors
The 25 scholars most cited alongside Torsten Exner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1998 | 171 | |
| 2 | 1999 | 105 | |
| 3 | 1998 | 52 | |
| 4 | 1999 | 38 | |
| 5 | 1996 | 34 | |
| 6 | 1997 | 25 | |
| 7 | 1999 | 19 | |
| 8 | 1996 | 15 | |
| 9 | 1998 | 1 |
About Torsten Exner
Torsten Exner is a scholar working on Molecular Biology, Cell Biology, Nuclear Energy and Engineering, Waste Management and Disposal and Infectious Diseases, having authored 9 papers that have together received 460 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (6 papers), Protein Kinase Regulation and GTPase Signaling (4 papers), PI3K/AKT/mTOR signaling in cancer (2 papers), Cellular transport and secretion (1 paper) and Ion channel regulation and function (1 paper). The work is most often cited by research in Molecular Biology (320 citations), Cellular and Molecular Neuroscience (50 citations), Physiology (12 citations), Cell Biology (32 citations) and Sensory Systems (12 citations). Torsten Exner has collaborated with scholars based in Germany, France and Switzerland. Frequent co-authors include Bernd Nürnberg, Udo Maier, Daniela Leopoldt, Theodor Hanck, Reinhard Wetzker, Patricia Viard, Jean Mironneau, Nathalie Macrez, Theo Schäfer and Reinhard Jahn. Their work appears in journals such as Biochemical Journal, Journal of Medicinal Chemistry, European Journal of Biochemistry, Analytical Biochemistry and The FASEB Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.