Thomas Haaf
Impact in
- Sensory Systems top 5%
- Olfactory and Sensory Function Studies
-
- Genomics and Chromatin Dynamics
- RNA Research and Splicing
- RNA and protein synthesis mechanisms
- DNA Repair Mechanisms
- RNA modifications and cancer
Papers in
-
- Genomics and Chromatin Dynamics 2
- RNA and protein synthesis mechanisms 2
- DNA Repair Mechanisms 1
- CRISPR and Genetic Engineering 1
- Co-authors
- Michael C. Schmid (1 shared paper)Michael Schmid (2 shared papers)D. L. Hayman (1 shared paper)Gustavo Glusman (1 shared paper)Miriam Khen (1 shared paper)Doron Lancet (1 shared paper)Dror Sharon (1 shared paper)Yitzhak Pilpel (1 shared paper)
- Journals
- Experimental Cell Research (2 papers)Nature Genetics (1 paper)Genomics (1 paper)American Journal of Medical Genetics (1 paper)
- Partner nations
- GermanyUnited StatesAustralia
In The Last Decade
Thomas Haaf
5 papers receiving 398 citations
Peers
Comparison fields: 5 of 46
- Sensory Systems 54
- Molecular Biology 306
- Nutrition and Dietetics 50
- Genetics 92
- Cellular and Molecular Neuroscience 59
Countries citing papers authored by Thomas Haaf
This map shows the geographic impact of Thomas Haaf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Haaf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Haaf more than expected).
Fields of papers citing papers by Thomas Haaf
This network shows the impact of papers produced by Thomas Haaf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Haaf. The network helps show where Thomas Haaf may publish in the future.
Co-authors
The 11 scholars most cited alongside Thomas Haaf, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1991 | 173 | |
| 2 | 1991 | 97 | |
| 3 | 1999 | 91 | |
| 4 | 1996 | 30 | |
| 5 | 1991 | 11 |
About Thomas Haaf
Thomas Haaf is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Pediatrics, Perinatology and Child Health, Sensory Systems and Nutrition and Dietetics, having authored 5 papers that have together received 402 indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (2 papers), RNA and protein synthesis mechanisms (2 papers), Genetics and Neurodevelopmental Disorders (1 paper), Fetal and Pediatric Neurological Disorders (1 paper), Pharmacological Effects and Toxicity Studies (1 paper), DNA Repair Mechanisms (1 paper), CRISPR and Genetic Engineering (1 paper) and Chromosomal and Genetic Variations (1 paper). The work is most often cited by research in Sensory Systems (54 citations), Molecular Biology (306 citations), Nutrition and Dietetics (50 citations), Genetics (92 citations) and Cellular and Molecular Neuroscience (59 citations). Thomas Haaf has collaborated with scholars based in Germany, United States and Australia. Frequent co-authors include Michael C. Schmid, Michael Schmid, D. L. Hayman, Gustavo Glusman, Miriam Khen, Doron Lancet, Dror Sharon, Yitzhak Pilpel, Kenneth K. Kídd and David C. Ward. Their work appears in journals such as Experimental Cell Research, Nature Genetics, Genomics and American Journal of Medical Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.