Su Ran Mun
Impact in
- Cell Biology top 10%
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
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- Autophagy in Disease and Therapy
Papers in
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- Autophagy in Disease and Therapy 7
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- Ubiquitin and proteasome pathways 4
- Protein Degradation and Inhibitors 2
- Signaling Pathways in Disease 1
- Histone Deacetylase Inhibitors Research 1
- Co-authors
- Yong Tae Kwon (11 shared papers)Chang Hoon Ji (5 shared papers)Bo Yeon Kim (5 shared papers)Aaron Ciechanover (4 shared papers)Ki Woon Sung (6 shared papers)Ah Jung Heo (3 shared papers)Hyunjoo Cha‐Molstad (3 shared papers)Su Hyun Lee (2 shared papers)
- Journals
- Autophagy (1 paper)Nature Communications (1 paper)Journal of Cell Science (1 paper)Molecular Neurodegeneration (1 paper)Molecular Cell (1 paper)
- Partner nations
- South KoreaUnited StatesPuerto Rico
In The Last Decade
Su Ran Mun
11 papers receiving 417 citations
Peers
Comparison fields: 5 of 54
- Cell Biology 117
- Epidemiology 200
- Physiology 21
- Molecular Biology 278
- Parasitology 25
Countries citing papers authored by Su Ran Mun
This map shows the geographic impact of Su Ran Mun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Su Ran Mun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Su Ran Mun more than expected).
Fields of papers citing papers by Su Ran Mun
This network shows the impact of papers produced by Su Ran Mun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Su Ran Mun. The network helps show where Su Ran Mun may publish in the future.
Co-authors
The 25 scholars most cited alongside Su Ran Mun, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 108 | |
| 2 | 2018 | 78 | |
| 3 | 2018 | 59 | |
| 4 | 2023 | 48 | |
| 5 | 2018 | 41 | |
| 6 | 2016 | 31 | |
| 7 | 2022 | 20 | |
| 8 | 2023 | 13 | |
| 9 | 2018 | 12 | |
| 10 | 2022 | 6 | |
| 11 | 2024 | 4 |
About Su Ran Mun
Su Ran Mun is a scholar working on Epidemiology, Molecular Biology, Cell Biology, Neurology and Pulmonary and Respiratory Medicine, having authored 11 papers that have together received 420 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (7 papers), Ubiquitin and proteasome pathways (4 papers), Protein Degradation and Inhibitors (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Signaling Pathways in Disease (1 paper), Prostate Cancer Treatment and Research (1 paper), Cancer, Hypoxia, and Metabolism (1 paper) and Histone Deacetylase Inhibitors Research (1 paper). The work is most often cited by research in Cell Biology (117 citations), Epidemiology (200 citations), Physiology (21 citations), Molecular Biology (278 citations) and Parasitology (25 citations). Su Ran Mun has collaborated with scholars based in South Korea, United States and Puerto Rico. Frequent co-authors include Yong Tae Kwon, Chang Hoon Ji, Bo Yeon Kim, Aaron Ciechanover, Ki Woon Sung, Ah Jung Heo, Hyunjoo Cha‐Molstad, Su Hyun Lee, Joonsung Hwang and Min Ju Lee. Their work appears in journals such as Autophagy, Nature Communications, Journal of Cell Science, Molecular Neurodegeneration and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.