Stefan Kins

4.0k citations
72 papers · 3.0k · h-index 32

Impact in

Papers in

    • Alzheimer's disease research and treatments 48
    • Prion Diseases and Protein Misfolding 9
    • Amyloidosis: Diagnosis, Treatment, Outcomes 6
    • Ubiquitin and proteasome pathways 5

Stefan Kins

72 papers receiving 3.0k citations

Peers

Stefan Kins
Comparison fields: 5 of 103
  • Physiology 1.6k
  • Cellular and Molecular Neuroscience 974
  • Cell Biology 773
  • Developmental Neuroscience 111
  • Neurology 211
Replace Ruth G. Perez with:
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Citations per field
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Citations per year

Countries citing papers authored by Stefan Kins

Since Specialization
Citations

This map shows the geographic impact of Stefan Kins's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan Kins with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan Kins more than expected).

Fields of papers citing papers by Stefan Kins

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan Kins. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan Kins. The network helps show where Stefan Kins may publish in the future.

Co-authors

The 25 scholars most cited alongside Stefan Kins, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Stefan Kins Line = papers co-authored together Stefan Kins links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 72 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005240
2 2000230
3 2005180
4 2001172
5 2002157
6 2003136
7 2006101
8 200998
9 201488
10 201175
11 200069
12 200866
13 200665
14 201965
15 201464
16 201162
17 201757
18 201754
19 201446
20 200246

About Stefan Kins

Stefan Kins is a scholar working on Physiology, Molecular Biology, Cell Biology, Cellular and Molecular Neuroscience and Nutrition and Dietetics, having authored 72 papers that have together received 3.0k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (48 papers), Cellular transport and secretion (19 papers), Neuroscience and Neuropharmacology Research (15 papers), Prion Diseases and Protein Misfolding (9 papers), Endoplasmic Reticulum Stress and Disease (8 papers), Microtubule and mitosis dynamics (7 papers), Amyloidosis: Diagnosis, Treatment, Outcomes (6 papers) and Ubiquitin and proteasome pathways (5 papers). The work is most often cited by research in Physiology (1.6k citations), Cellular and Molecular Neuroscience (974 citations), Cell Biology (773 citations), Developmental Neuroscience (111 citations) and Neurology (211 citations). Stefan Kins has collaborated with scholars based in Germany, United States and Switzerland. Frequent co-authors include Joachim Kirsch, Heinrich Betz, Jürgen Götz, Roger M. Nitsch, Simone Eggert, Ulrike Müller, Konrad Beyreuther, Gerardo Morfini, Katja Wagner and Anita Szodorai. Their work appears in journals such as Biological Chemistry, Journal of Neuroscience, Frontiers in Molecular Neuroscience, European Journal of Neuroscience and Journal of Neurochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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