Sonja Aits
Impact in
- Physiology top 1%
- Calcium signaling and nucleotide metabolism
- Lysosomal Storage Disorders Research
- Epidemiology top 5%
- Autophagy in Disease and Therapy
Papers in
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- Cell death mechanisms and regulation 2
- RNA modifications and cancer 1
- Epigenetics and DNA Methylation 1
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- Autophagy in Disease and Therapy 7
- Co-authors
- Marja Jäättelä (5 shared papers)Jesper Nylandsted (2 shared papers)Catharina Svanborg (7 shared papers)Elisabeth Corcelle–Termeau (2 shared papers)Lotta Gustafsson (5 shared papers)Jennifer Kricker (1 shared paper)Siri Tvingsholm (1 shared paper)Thomas Farkas (1 shared paper)
In The Last Decade
Sonja Aits
15 papers receiving 1.3k citations
Sonja Aits's Hit Papers
Peers
Comparison fields: 5 of 107
- Physiology 211
- Epidemiology 412
- Cell Biology 199
- Molecular Biology 661
- Parasitology 60
Countries citing papers authored by Sonja Aits
This map shows the geographic impact of Sonja Aits's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sonja Aits with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sonja Aits more than expected).
Fields of papers citing papers by Sonja Aits
This network shows the impact of papers produced by Sonja Aits. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sonja Aits. The network helps show where Sonja Aits may publish in the future.
Co-authors
The 25 scholars most cited alongside Sonja Aits, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Lysosomal cell death at a glance Hit paper breakdown → | 2013 | 527 |
| 2 | 2015 | 319 | |
| 3 | 2015 | 77 | |
| 4 | 2007 | 65 | |
| 5 | 2008 | 62 | |
| 6 | 2012 | 61 | |
| 7 | 2009 | 55 | |
| 8 | 2011 | 38 | |
| 9 | 2008 | 36 | |
| 10 | 2011 | 28 | |
| 11 | 2016 | 18 | |
| 12 | 2008 | 18 | |
| 13 | 2019 | 15 | |
| 14 | 2022 | 3 | |
| 15 | 2009 | 2 | |
| 16 | 2023 | 1 |
About Sonja Aits
Sonja Aits is a scholar working on Molecular Biology, Epidemiology, Cell Biology, Physiology and Oncology, having authored 16 papers that have together received 1.3k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (7 papers), Calcium signaling and nucleotide metabolism (4 papers), Cellular transport and secretion (3 papers), Cell death mechanisms and regulation (2 papers), Lysosomal Storage Disorders Research (2 papers), Cancer Research and Treatments (2 papers), RNA modifications and cancer (1 paper) and Epigenetics and DNA Methylation (1 paper). The work is most often cited by research in Physiology (211 citations), Epidemiology (412 citations), Cell Biology (199 citations), Molecular Biology (661 citations) and Parasitology (60 citations). Sonja Aits has collaborated with scholars based in Sweden, Singapore and Denmark. Frequent co-authors include Marja Jäättelä, Jesper Nylandsted, Catharina Svanborg, Elisabeth Corcelle–Termeau, Lotta Gustafsson, Jennifer Kricker, Siri Tvingsholm, Thomas Farkas, Anne‐Marie Ellegaard and Monika Mortensen. Their work appears in journals such as PLoS ONE, Assay and Drug Development Technologies, Advances in experimental medicine and biology, International Journal of Cancer and Autophagy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.