Simone Orlando
Impact in
- Immunology and Allergy top 10%
- Cell Adhesion Molecules Research
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- Immune Response and Inflammation
- Immune Cell Function and Interaction
- Immunotherapy and Immune Responses
Papers in
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- Immune Response and Inflammation 4
- Toxin Mechanisms and Immunotoxins 1
- Immunotherapy and Immune Responses 1
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- Cell Adhesion Molecules Research 3
- Co-authors
- Alberto Mantovani (4 shared papers)Francesco Blasi (2 shared papers)Massimo Resnati (2 shared papers)Marina Sironi (2 shared papers)Sergio Bernasconi (2 shared papers)Shafaat A. Rabbani (1 shared paper)Bernard Degryse (1 shared paper)Giuseppe Bianchi (1 shared paper)
- Journals
- Cytokine (1 paper)The Journal of Immunology (1 paper)Oncogene (1 paper)Proceedings of the National Academy of Sciences (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- ItalyCanadaUnited States
In The Last Decade
Simone Orlando
6 papers receiving 387 citations
Peers
Comparison fields: 5 of 68
- Immunology and Allergy 65
- Immunology 196
- Cancer Research 130
- Hematology 52
- Oncology 78
Countries citing papers authored by Simone Orlando
This map shows the geographic impact of Simone Orlando's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simone Orlando with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simone Orlando more than expected).
Fields of papers citing papers by Simone Orlando
This network shows the impact of papers produced by Simone Orlando. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simone Orlando. The network helps show where Simone Orlando may publish in the future.
Co-authors
The 22 scholars most cited alongside Simone Orlando, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1997 | 95 | |
| 2 | 2000 | 95 | |
| 3 | 2001 | 82 | |
| 4 | 2004 | 61 | |
| 5 | 1999 | 56 | |
| 6 | 2000 | 12 |
About Simone Orlando
Simone Orlando is a scholar working on Immunology, Immunology and Allergy, Cancer Research, Oncology and Molecular Biology, having authored 6 papers that have together received 401 indexed citations. Recurring topics across this work include Immune Response and Inflammation (4 papers), Cell Adhesion Molecules Research (3 papers), Protease and Inhibitor Mechanisms (2 papers), Chemokine receptors and signaling (2 papers), Inflammasome and immune disorders (1 paper), NF-κB Signaling Pathways (1 paper), Toxin Mechanisms and Immunotoxins (1 paper) and Immunotherapy and Immune Responses (1 paper). The work is most often cited by research in Immunology and Allergy (65 citations), Immunology (196 citations), Cancer Research (130 citations), Hematology (52 citations) and Oncology (78 citations). Simone Orlando has collaborated with scholars based in Italy, Canada and United States. Frequent co-authors include Alberto Mantovani, Francesco Blasi, Massimo Resnati, Marina Sironi, Sergio Bernasconi, Shafaat A. Rabbani, Bernard Degryse, Giuseppe Bianchi, Diana Boraschi and Alan H. Drummond. Their work appears in journals such as Cytokine, The Journal of Immunology, Oncogene, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.