Sho‐Ya Wang

3.0k citations

55 papers · 2.5k · h-index 29

Impact in

Sensory Systems top 1%
- Ion Channels and Receptors
Cellular and Molecular Neuroscience top 2%
- Neuroscience and Neuropharmacology Research
- Neuroscience and Neural Engineering

Papers in

Molecular Biology 52
- Ion channel regulation and function 43
- Heat shock proteins research 4
- Nicotinic Acetylcholine Receptors Study 4
- Receptor Mechanisms and Signaling 3
Cellular and Molecular Neuroscience 34
- Neuroscience and Neuropharmacology Research 27
- Neuroscience and Neural Engineering 8

Co-authors: Ging Kuo Wang (44 shared papers)Carla Nau (7 shared papers)Lorraine J. Gudas (4 shared papers)Gregory J. LaRosa (1 shared paper)Jane Mitchell (6 shared papers)Gary R. Strichartz (3 shared papers)David L. Williams (1 shared paper)John O’Reilly (3 shared papers)
Journals: Molecular Pharmacology (10 papers)Biophysical Journal (6 papers)The Journal of Physiology (4 papers)Proceedings of the National Academy of Sciences (3 papers)The Journal of General Physiology (3 papers)
Partner nations: United States Germany Czechia

In The Last Decade

Sho‐Ya Wang

55 papers receiving 2.5k citations

Peers

Countries citing papers authored by Sho‐Ya Wang

Since Specialization

Citations

This map shows the geographic impact of Sho‐Ya Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sho‐Ya Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sho‐Ya Wang more than expected).

Fields of papers citing papers by Sho‐Ya Wang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sho‐Ya Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sho‐Ya Wang. The network helps show where Sho‐Ya Wang may publish in the future.

Co-authors

The 25 scholars most cited alongside Sho‐Ya Wang, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Sho‐Ya Wang Line = papers co-authored together Sho‐Ya Wang links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 55 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Voltage-gated sodium channels as primary targets of diverse lipid-soluble neurotoxins Cellular Signalling ·Sho‐Ya Wang	2002	238
2	Molecular cloning of gene sequences transcriptionally regulated by retinoic acid and dibutyryl cyclic AMP in cultured mouse teratocarcinoma cells Developmental Biology ·Sho‐Ya Wang, Gregory J. LaRosa, Lorraine J. Gudas	1985	198
3	PKA/AKAP/VR-1 Module: A Common Link of G_s-Mediated Signaling to Thermal Hyperalgesia Journal of Neuroscience ·P. K. Rathee, Carsten Distler, Otilia Obreja, Winfried Neuhuber, Ging Kuo Wang, Sho‐Ya Wang, Carla Nau, Michaela Kress	2002	193
4	Single point mutations affect fatty acid block of human myocardial sodium channel α subunit Na ⁺ channels Proceedings of the National Academy of Sciences ·Yong‐Fu Xiao, Qingen Ke, Sho‐Ya Wang, Klaus Auktor, Yinke Yang, Ging Kuo Wang, James P. Morgan, Alexander Leaf	2001	108
5	State-dependent block of voltage-gated Na+ channels by amitriptyline via the local anesthetic receptor and its implication for neuropathic pain Pain ·Ging Kuo Wang, Corinna Russell, Sho‐Ya Wang	2004	100
6	Point mutations in segment I-S6 render voltage-gated Na ⁺ channels resistant to batrachotoxin Proceedings of the National Academy of Sciences ·Sho‐Ya Wang, Ging Kuo Wang	1998	96
7	Residues in Na+ Channel D3-S6 Segment Modulate both Batrachotoxin and Local Anesthetic Affinities Biophysical Journal ·Sho‐Ya Wang, Carla Nau, Ging Kuo Wang	2000	92
8	Point Mutations at N434 in D1-S6 of μ1 Na⁺Channels Modulate Binding Affinity and Stereoselectivity of Local Anesthetic Enantiomers Molecular Pharmacology ·Carla Nau, Sho‐Ya Wang, Gary R. Strichartz, Ging Kuo Wang	1999	89
9	Batrachotoxin-Resistant Na+ Channels Derived from Point Mutations in Transmembrane Segment D4-S6 Biophysical Journal ·Sho‐Ya Wang, Ging Kuo Wang	1999	77
10	Identification, purification, and characterization of two distinct avian vitellogenins Biochemistry ·Sho‐Ya Wang, David L. Williams	1980	76
11	State- and Use-Dependent Block of Muscle Nav1.4 and Neuronal Nav1.7 Voltage-Gated Na+ Channel Isoforms by Ranolazine Molecular Pharmacology ·Ging Kuo Wang, Joanna Calderon, Sho‐Ya Wang	2007	70
12	A tyrosine residue in TM6 of the Vanilloid Receptor TRPV1 involved in desensitization and calcium permeability of capsaicin-activated currents Molecular and Cellular Neuroscience ·Durga P. Mohapatra, Sho‐Ya Wang, Ging Kuo Wang, Carla Nau	2003	66
13	Block of Neuronal Na+Channels by Antidepressant Duloxetine in a State-dependent Manner Anesthesiology ·Sho‐Ya Wang, Joanna Calderon, Ging Kuo Wang	2010	65
14	Tryptophan Scanning of D1S6 and D4S6 C-Termini in Voltage-Gated Sodium Channels Biophysical Journal ·Sho‐Ya Wang, Kaitlin Bonner, Corinna Russell, Ging Kuo Wang	2003	59
15	Block of Human Heart hH1 Sodium Channels by the Enantiomers of Bupivacaine Anesthesiology ·Carla Nau, Sho‐Ya Wang, Gary R. Strichartz, Ging Kuo Wang	2000	55
16	Disparate Role of Na⁺Channel D2-S6 Residues in Batrachotoxin and Local Anesthetic Action Molecular Pharmacology ·Sho‐Ya Wang, Maria Barile, Ging Kuo Wang	2001	55
17	Mexiletine block of wild‐type and inactivation‐deficient human skeletal muscle hNav1.4 Na⁺ channels The Journal of Physiology ·Ging Kuo Wang, Corinna Russell, Sho‐Ya Wang	2003	51
18	How Batrachotoxin Modifies the Sodium Channel Permeation Pathway: Computer Modeling and Site-Directed Mutagenesis Molecular Pharmacology ·Sho‐Ya Wang, Jane Mitchell, Denis B. Tikhonov, Boris S. Zhorov, Ging Kuo Wang	2005	50
19	Residue-Specific Effects on Slow Inactivation at V787 in D2-S6 of Nav1.4 Sodium Channels Biophysical Journal ·John O’Reilly, Sho‐Ya Wang, Ging Kuo Wang	2001	47
20	Block of Inactivation-deficient Na+ Channels by Local Anesthetics in Stably Transfected Mammalian Cells The Journal of General Physiology ·Sho‐Ya Wang, Jane Mitchell, Edward Moczydlowski, Ging Kuo Wang	2004	46

About Sho‐Ya Wang

Sho‐Ya Wang is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Cardiology and Cardiovascular Medicine, Physiology and Sensory Systems, having authored 55 papers that have together received 2.5k indexed citations. Recurring topics across this work include Ion channel regulation and function (43 papers), Neuroscience and Neuropharmacology Research (27 papers), Cardiac electrophysiology and arrhythmias (26 papers), Pain Mechanisms and Treatments (8 papers), Neuroscience and Neural Engineering (8 papers), Heat shock proteins research (4 papers), Nicotinic Acetylcholine Receptors Study (4 papers) and Receptor Mechanisms and Signaling (3 papers). The work is most often cited by research in Sensory Systems (276 citations), Cellular and Molecular Neuroscience (988 citations), Cardiology and Cardiovascular Medicine (570 citations), Molecular Biology (1.7k citations) and Physiology (417 citations). Sho‐Ya Wang has collaborated with scholars based in United States, Germany and Czechia. Frequent co-authors include Ging Kuo Wang, Carla Nau, Lorraine J. Gudas, Gregory J. LaRosa, Jane Mitchell, Gary R. Strichartz, David L. Williams, John O’Reilly, Michaela Kress and Winfried Neuhuber. Their work appears in journals such as Molecular Pharmacology, Biophysical Journal, The Journal of Physiology, Proceedings of the National Academy of Sciences and The Journal of General Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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