Samuel Burnim

859 citations
10 papers · 397 · h-index 10

Impact in

Papers in

    • Retinal Development and Disorders 3
    • Fibroblast Growth Factor Research 2
    • Peroxisome Proliferator-Activated Receptors 1
    • Retinal Diseases and Treatments 3

Samuel Burnim

10 papers receiving 393 citations

Peers

Samuel Burnim
Comparison fields: 5 of 69
  • Ophthalmology 141
  • Radiology, Nuclear Medicine and Imaging 90
  • Neurology 30
  • Biochemistry 26
  • Molecular Biology 181
Replace Tianwei Ellen Zhou with:
Tianwei Ellen Zhou Canada
William S. Wright United States
L. Goodwin Burgess United States
Patricia M. Notario United States
Sheila Pons-Vázquez Spain
Carmel McVicar United Kingdom
Siqi Wu United States
Linling Zhu China
Dilani Siriwardena United Kingdom
Daisuke Shiba Japan
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Citations per field
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Citations per year

Countries citing papers authored by Samuel Burnim

Since Specialization
Citations

This map shows the geographic impact of Samuel Burnim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samuel Burnim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samuel Burnim more than expected).

Fields of papers citing papers by Samuel Burnim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Samuel Burnim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samuel Burnim. The network helps show where Samuel Burnim may publish in the future.

Co-authors

The 25 scholars most cited alongside Samuel Burnim, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Samuel Burnim Line = papers co-authored together Samuel Burnim links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 201766
2 201863
3 201755
4 201652
5 201640
6 201937
7 201729
8 201720
9 201718
10 201617

About Samuel Burnim

Samuel Burnim is a scholar working on Molecular Biology, Ophthalmology, Radiology, Nuclear Medicine and Imaging, Biochemistry and Immunology, having authored 10 papers that have together received 397 indexed citations. Recurring topics across this work include Retinopathy of Prematurity Studies (3 papers), Retinal Diseases and Treatments (3 papers), Retinal Development and Disorders (3 papers), Eicosanoids and Hypertension Pharmacology (2 papers), Phagocytosis and Immune Regulation (2 papers), Fibroblast Growth Factor Research (2 papers), Axon Guidance and Neuronal Signaling (1 paper) and Peroxisome Proliferator-Activated Receptors (1 paper). The work is most often cited by research in Ophthalmology (141 citations), Radiology, Nuclear Medicine and Imaging (90 citations), Neurology (30 citations), Biochemistry (26 citations) and Molecular Biology (181 citations). Samuel Burnim has collaborated with scholars based in United States, Sweden and Hong Kong. Frequent co-authors include Steven Meng, Lois E. H. Smith, Chi‐Hsiu Liu, Zhongxiao Wang, Yan Gong, Raffael Liegl, Zhongjie Fu, Ye Sun, Ann Hellström and Thomas Fredrick. Their work appears in journals such as EBioMedicine, The FASEB Journal, Arteriosclerosis Thrombosis and Vascular Biology, Diabetes and Cell Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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