Robert P. Fordham

7 papers receiving 972 citations

Robert P. Fordham's Hit Papers

YAP/TAZ-Dependent Reprogramming of Colonic Epithelium Links ECM Remodeling to Tissue Regeneration 2017 · 511 citations
5110+3+6Years since publication100200300400500

Peers

Robert P. Fordham
Comparison fields: 5 of 73
  • Oncology 439
  • Cell Biology 207
  • Genetics 215
  • Hepatology 45
  • Molecular Biology 402
Replace Hjalte List Larsen with:
Hjalte List Larsen Denmark
Jessica H. Geahlen United States
Miriam Gordillo United States
Hiroo Ueno Japan
Nicolas Chuvin France
Fanny Jaulin France
Matthew R. LeBoeuf United States
James E. Annis United States
Maryam Yousefi United States
Zahra Kabiri United States
Robert P. Fordham relative to Hjalte List Larsen Denmark Hjalte List Larsen's profile →
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Citations per year

Countries citing papers authored by Robert P. Fordham

Since Specialization
Citations

This map shows the geographic impact of Robert P. Fordham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert P. Fordham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert P. Fordham more than expected).

Fields of papers citing papers by Robert P. Fordham

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert P. Fordham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert P. Fordham. The network helps show where Robert P. Fordham may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert P. Fordham, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert P. Fordham Line = papers co-authored together Robert P. Fordham links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1
YAP/TAZ-Dependent Reprogramming of Colonic Epithelium Links ECM Remodeling to Tissue Regeneration
Hit paper breakdown →
2017511
2 2013342
3 201375
4 201026
5 202118
6 20123
7 20152

About Robert P. Fordham

Robert P. Fordham is a scholar working on Cell Biology, Oncology, Urology, Dermatology and Molecular Biology, having authored 7 papers that have together received 977 indexed citations. Recurring topics across this work include Cancer Cells and Metastasis (2 papers), Pluripotent Stem Cells Research (2 papers), CRISPR and Genetic Engineering (1 paper), Pancreatic function and diabetes (1 paper), Congenital Diaphragmatic Hernia Studies (1 paper), TGF-β signaling in diseases (1 paper), Hair Growth and Disorders (1 paper) and Cancer and Skin Lesions (1 paper). The work is most often cited by research in Oncology (439 citations), Cell Biology (207 citations), Genetics (215 citations), Hepatology (45 citations) and Molecular Biology (402 citations). Robert P. Fordham has collaborated with scholars based in United Kingdom, Japan and Denmark. Frequent co-authors include Kim Bak Jensen, Pawel J. Schweiger, Tetsuya Nakamura, Mamoru Watanabe, Ole Haagen Nielsen, Shiro Yui, Nicholas R. F. Hannan, Ludovic Vallier, Christoffer Soendergaard and Roger A. Pedersen. Their work appears in journals such as Cell stem cell, Stem Cell Reports, Nature Communications, Stem Cells and Development and The Journal of Experimental Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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