Robert N. Cox

435 citations
11 papers · 381 · h-index 10

Impact in

Papers in

    • Nicotinic Acetylcholine Receptors Study 4
    • Ion channel regulation and function 3
    • Receptor Mechanisms and Signaling 3
    • Muscle Physiology and Disorders 1
    • Cardiomyopathy and Myosin Studies 5
    • Cardiac electrophysiology and arrhythmias 1
    • Cardiovascular Effects of Exercise 1

Robert N. Cox

11 papers receiving 338 citations

Peers

Robert N. Cox
Comparison fields: 5 of 46
  • Cardiology and Cardiovascular Medicine 213
  • Cellular and Molecular Neuroscience 71
  • Molecular Biology 267
  • Cell Biology 47
  • Biomedical Engineering 98
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Citations per year

Countries citing papers authored by Robert N. Cox

Since Specialization
Citations

This map shows the geographic impact of Robert N. Cox's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert N. Cox with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert N. Cox more than expected).

Fields of papers citing papers by Robert N. Cox

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert N. Cox. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert N. Cox. The network helps show where Robert N. Cox may publish in the future.

Co-authors

The 11 scholars most cited alongside Robert N. Cox, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert N. Cox Line = papers co-authored together Robert N. Cox links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 1982130
2 198084
3 198549
4 198126
5 198326
6 198418
7 198113
8 197911
9 198611
10 197910
11 19843

About Robert N. Cox

Robert N. Cox is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Biomedical Engineering, Cellular and Molecular Neuroscience and Organic Chemistry, having authored 11 papers that have together received 381 indexed citations. Recurring topics across this work include Cardiomyopathy and Myosin Studies (5 papers), Nicotinic Acetylcholine Receptors Study (4 papers), Ion channel regulation and function (3 papers), Receptor Mechanisms and Signaling (3 papers), Muscle activation and electromyography studies (2 papers), Cardiac electrophysiology and arrhythmias (1 paper), Cardiovascular Effects of Exercise (1 paper) and Muscle Physiology and Disorders (1 paper). The work is most often cited by research in Cardiology and Cardiovascular Medicine (213 citations), Cellular and Molecular Neuroscience (71 citations), Molecular Biology (267 citations), Cell Biology (47 citations) and Biomedical Engineering (98 citations). Robert N. Cox has collaborated with scholars based in United States and Russia. Frequent co-authors include Philip W. Brandt, Masataka Kawai, Thomas F. Robinson, Arthur Karlin, Mario DiPaola, Peter Lobel, E Holtzman, Richard Hudson, Eric Holtzman and Peter N. Kao. Their work appears in journals such as The Journal of Membrane Biology, Analytical Biochemistry, Cold Spring Harbor Symposia on Quantitative Biology, Journal of Biological Chemistry and Journal of Muscle Research and Cell Motility.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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