Robert J. Ife

2.2k citations
53 papers · 1.8k · h-index 24

Impact in

Papers in

    • Ion Transport and Channel Regulation 10
    • Receptor Mechanisms and Signaling 8
    • ATP Synthase and ATPases Research 8
    • Protein Kinase Regulation and GTPase Signaling 6
    • Chemical Synthesis and Analysis 5
    • Enzyme function and inhibition 5

Robert J. Ife

52 papers receiving 1.7k citations

Peers

Robert J. Ife
Comparison fields: 5 of 101
  • Organic Chemistry 446
  • Gastroenterology 69
  • Molecular Biology 880
  • Cancer Research 173
  • Oncology 235
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Citations per field
00.5×4.1×
R. Michael Lawrence · 1×
Citations per year

Countries citing papers authored by Robert J. Ife

Since Specialization
Citations

This map shows the geographic impact of Robert J. Ife's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Ife with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Ife more than expected).

Fields of papers citing papers by Robert J. Ife

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Ife. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Ife. The network helps show where Robert J. Ife may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert J. Ife, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert J. Ife Line = papers co-authored together Robert J. Ife links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 53 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1999297
2 2001147
3 2003100
4 200398
5 200397
6 200375
7 199973
8 198565
9 200260
10 199248
11 199047
12 199145
13 200041
14 200140
15 199239
16 199537
17 200135
18 200234
19 198632
20 197632

About Robert J. Ife

Robert J. Ife is a scholar working on Molecular Biology, Organic Chemistry, Oncology, Immunology and Surgery, having authored 53 papers that have together received 1.8k indexed citations. Recurring topics across this work include Ion Transport and Channel Regulation (10 papers), Receptor Mechanisms and Signaling (8 papers), ATP Synthase and ATPases Research (8 papers), Protein Kinase Regulation and GTPase Signaling (6 papers), Mast cells and histamine (6 papers), Drug Transport and Resistance Mechanisms (5 papers), Chemical Synthesis and Analysis (5 papers) and Enzyme function and inhibition (5 papers). The work is most often cited by research in Organic Chemistry (446 citations), Gastroenterology (69 citations), Molecular Biology (880 citations), Cancer Research (173 citations) and Oncology (235 citations). Robert J. Ife has collaborated with scholars based in United Kingdom, United States and Canada. Frequent co-authors include Colin A. Leach, Deirdre M. B. Hickey, Kevin J. Milliner, Kitty Moores, David J. Keeling, Colin H. Macphee, David G. Tew, David G. Smith, Robert W. Ward and Helen Boyd. Their work appears in journals such as Bioorganic & Medicinal Chemistry Letters, Journal of Medicinal Chemistry, Biochemical Pharmacology, Journal of the Chemical Society Perkin Transactions 1 and European Journal of Medicinal Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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