Robert H. Eibl

29 papers receiving 1.3k citations

Peers

Robert H. Eibl
Comparison fields: 5 of 89
  • Genetics 644
  • Cancer Research 356
  • Oncology 458
  • Neurology 201
  • Cell Biology 165
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Citations per field
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Citations per year

Countries citing papers authored by Robert H. Eibl

Since Specialization
Citations

This map shows the geographic impact of Robert H. Eibl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert H. Eibl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert H. Eibl more than expected).

Fields of papers citing papers by Robert H. Eibl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert H. Eibl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert H. Eibl. The network helps show where Robert H. Eibl may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert H. Eibl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert H. Eibl Line = papers co-authored together Robert H. Eibl links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 30 papers — load more, or switch the sort, to bring in the rest.

#Work
1
p53 mutations are associated with 17p allelic loss in grade II and grade III astrocytoma.
1992257
2 1993205
3
p53 mutations in nonastrocytic human brain tumors.
1991204
4 1993197
5 199579
6 200552
7
Activity of the DNA repair protein O6-methylguanine-DNA methyltransferase in human tumor and corresponding normal tissue.
199651
8 199549
9 202146
10
A model for primitive neuroectodermal tumors in transgenic neural transplants harboring the SV40 large T antigen.
199433
11 199128
12
Oncogene complementation in fetal brain transplants.
199228
13 202325
14 200524
15 199323
16 199418
17 200416
18 202214
19 199014
20 199213

About Robert H. Eibl

Robert H. Eibl is a scholar working on Molecular Biology, Genetics, Atomic and Molecular Physics, and Optics, Oncology and Cell Biology, having authored 30 papers that have together received 1.4k indexed citations. Recurring topics across this work include Glioma Diagnosis and Treatment (8 papers), Cancer Genomics and Diagnostics (6 papers), Force Microscopy Techniques and Applications (5 papers), Cancer-related Molecular Pathways (4 papers), Mechanical and Optical Resonators (4 papers), Virus-based gene therapy research (4 papers), Microtubule and mitosis dynamics (3 papers) and Neuroblastoma Research and Treatments (3 papers). The work is most often cited by research in Genetics (644 citations), Cancer Research (356 citations), Oncology (458 citations), Neurology (201 citations) and Cell Biology (165 citations). Robert H. Eibl has collaborated with scholars based in Switzerland, Germany and United States. Frequent co-authors include Hiroko Ohgaki, OD Wiestler, Andreas von Deimling, Markus Schneemann, Bernd R. Seizinger, David N. Louis, Richard Y. Chung, Otmar D. Wiestler, M. Gazi Yaşargil and Paul Kleihues. Their work appears in journals such as Recent results in cancer research, Cancers, Journal of Neuro-Oncology, Journal of Neuropathology & Experimental Neurology and Neuropathology and Applied Neurobiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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