Robert H. Eibl

1.7k citations
33 papers · 1.4k · h-index 17

Impact in

Papers in

    • Glioma Diagnosis and Treatment 8
    • Virus-based gene therapy research 4
    • Cellular Mechanics and Interactions 4

Robert H. Eibl

32 papers receiving 1.4k citations

Peers

Robert H. Eibl
Comparison fields: 5 of 92
  • Genetics 659
  • Cancer Research 374
  • Oncology 457
  • Neurology 204
  • Cell Biology 172
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Citations per field
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Citations per year

Countries citing papers authored by Robert H. Eibl

Since Specialization
Citations

This map shows the geographic impact of Robert H. Eibl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert H. Eibl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert H. Eibl more than expected).

Fields of papers citing papers by Robert H. Eibl

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert H. Eibl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert H. Eibl. The network helps show where Robert H. Eibl may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert H. Eibl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert H. Eibl Line = papers co-authored together Robert H. Eibl links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.

#Work
1
p53 mutations are associated with 17p allelic loss in grade II and grade III astrocytoma.
1992257
2
p53 mutations in nonastrocytic human brain tumors.
1991208
3 1993203
4 1993196
5 199579
6 200552
7 202151
8
Activity of the DNA repair protein O6-methylguanine-DNA methyltransferase in human tumor and corresponding normal tissue.
199650
9 199549
10
A model for primitive neuroectodermal tumors in transgenic neural transplants harboring the SV40 large T antigen.
199435
11
Oncogene complementation in fetal brain transplants.
199228
12 202327
13 199127
14 202025
15 200524
16 199323
17 199418
18 200417
19 202214
20 199213

About Robert H. Eibl

Robert H. Eibl is a scholar working on Genetics, Cell Biology, Cancer Research, Atomic and Molecular Physics, and Optics and Oncology, having authored 33 papers that have together received 1.4k indexed citations. Recurring topics across this work include Glioma Diagnosis and Treatment (8 papers), Cancer Genomics and Diagnostics (6 papers), Force Microscopy Techniques and Applications (6 papers), Cellular Mechanics and Interactions (4 papers), Mechanical and Optical Resonators (4 papers), Cancer-related Molecular Pathways (4 papers), Virus-based gene therapy research (4 papers) and CRISPR and Genetic Engineering (3 papers). The work is most often cited by research in Genetics (659 citations), Cancer Research (374 citations), Oncology (457 citations), Neurology (204 citations) and Cell Biology (172 citations). Robert H. Eibl has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Hiroko Ohgaki, OD Wiestler, Markus Schneemann, Andreas von Deimling, Iver Petersen, Richard Y. Chung, Bernd R. Seizinger, David N. Louis, Mahmut Gazi Yasargil and Otmar Dieter Wiestler. Their work appears in journals such as Recent results in cancer research, Cancers, Biochemical Society Transactions, Acta Neuropathologica and The Analyst.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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