Robert D. Terry

27.7k citations
174 papers · 24.2k · 10 hit papers · h-index 74

Impact in

  • Neurology top 0.1%
    • Neuroinflammation and Neurodegeneration Mechanisms
    • Parkinson's Disease Mechanisms and Treatments
  • Physiology top 0.05%
    • Alzheimer's disease research and treatments

Papers in

Robert D. Terry

170 papers receiving 22.1k citations

Robert D. Terry's Hit Papers

Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment 1991 · 3.7k citations
3.7k0+22+44Years since publication10002.0k3.0k

Peers

Robert D. Terry
Comparison fields: 5 of 180
  • Neurology 3.9k
  • Physiology 11.4k
  • Cellular and Molecular Neuroscience 7.0k
  • Psychiatry and Mental health 4.0k
  • Biological Psychiatry 656
Replace Lawrence A. Hansen with:
Lawrence A. Hansen United States
Allen D. Roses United States
Deborah C. Mash United States
Warren J. Strittmatter United States
Charles J. Duyckaerts France
D. E. Schmechel United States
Thomas Wısnıewskı United States
Stanley Isaac Rapoport United States
Robert Katzman United States
Jeffery M. Vance United States
Robert D. Terry relative to Lawrence A. Hansen United States Lawrence A. Hansen's profile →
Citations per field
00.5×1.5×
Lawrence A. Hansen · 1×
Citations per year

Countries citing papers authored by Robert D. Terry

Since Specialization
Citations

This map shows the geographic impact of Robert D. Terry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert D. Terry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert D. Terry more than expected).

Fields of papers citing papers by Robert D. Terry

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert D. Terry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert D. Terry. The network helps show where Robert D. Terry may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert D. Terry, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert D. Terry Line = papers co-authored together Robert D. Terry links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 174 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Physical basis of cognitive alterations in alzheimer's disease: Synapse loss is the major correlate of cognitive impairment
Hit paper breakdown →
19913671
2
Pathological verification of ischemic score in differentiation of dementias
Hit paper breakdown →
19801173
3
Clinical, pathological, and neurochemical changes in dementia: A subgroup with preserved mental status and numerous neocortical plaques
Hit paper breakdown →
19881009
4
Distribution of cholinergic neurons in rat brain: Demonstrated by the immunocytochemical localization of choline acetyltransferase
Hit paper breakdown →
1983902
5
Reduced somatostatin-like immunoreactivity in cerebral cortex from cases of Alzheimer disease and Alzheimer senile dementa
Hit paper breakdown →
1980799
6
Senile dementia of the Alzheimer type
Hit paper breakdown →
1983739
7
Some morphometric aspects of the brain in senile dementia of the alzheimer type
Hit paper breakdown →
1981725
8
Neocortical cell counts in normal human adult aging
Hit paper breakdown →
1987615
9
ULTRASTRUCTURAL STUDIES IN ALZHEIMER'S PRESENILE DEMENTIA.
Hit paper breakdown →
1964573
10 1991416
11 1994357
12 1963346
13 1992342
14
Alzheimer's disease : senile dementia and related disorders
1978340
15
Experiences with metastatic neoplasms involving the spinal cord
Hit paper breakdown →
1959339
16 1964333
17 1989331
18 1976323
19 1965306
20
Cortical and subcortical patterns of synaptophysinlike immunoreactivity in Alzheimer's disease.
1991271

About Robert D. Terry

Robert D. Terry is a scholar working on Physiology, Cellular and Molecular Neuroscience, Neurology, Neurology and Psychiatry and Mental health, having authored 174 papers that have together received 24.2k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (58 papers), Neuroscience and Neuropharmacology Research (20 papers), Prion Diseases and Protein Misfolding (16 papers), Dementia and Cognitive Impairment Research (15 papers), Cholinesterase and Neurodegenerative Diseases (15 papers), Memory and Neural Mechanisms (11 papers), Neurological diseases and metabolism (10 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). The work is most often cited by research in Neurology (3.9k citations), Physiology (11.4k citations), Cellular and Molecular Neuroscience (7.0k citations), Psychiatry and Mental health (4.0k citations) and Biological Psychiatry (656 citations). Robert D. Terry has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Richard M. DeTeresa, Eliezer Masliah, Lawrence A. Hansen, Robert Katzman, Robert Katzman, Arthur Peck, Nelson Butters, David P. Salmon, Robert Hill and Paula Altman Fuld. Their work appears in journals such as Journal of Neuropathology & Experimental Neurology, Annals of Neurology, Brain Research, Neurobiology of Aging and Acta Neuropathologica.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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