Ralf Egner
Impact in
- Cell Biology top 2%
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
- Epidemiology top 5%
- Autophagy in Disease and Therapy
Papers in
- Oncology 6
- Drug Transport and Resistance Mechanisms 5
-
- Cellular transport and secretion 4
- Endoplasmic Reticulum Stress and Disease 2
- Co-authors
- Karl Kuchler (8 shared papers)Michael Straub (2 shared papers)Detlef Wolf (2 shared papers)Michael Thumm (2 shared papers)Martin Schlumpberger (1 shared paper)Birgit Koch (1 shared paper)Marten Veenhuis (1 shared paper)Dieter H. Wolf (1 shared paper)
- Journals
- FEBS Letters (2 papers)Journal of Biological Chemistry (2 papers)Yeast (1 paper)Molecular and Cellular Biology (1 paper)Molecular Biology of the Cell (1 paper)
- Partner nations
- AustriaGermanySwitzerland
In The Last Decade
Ralf Egner
11 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 70
- Cell Biology 502
- Epidemiology 562
- Physiology 73
- Molecular Biology 654
- Oncology 197
Countries citing papers authored by Ralf Egner
This map shows the geographic impact of Ralf Egner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ralf Egner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ralf Egner more than expected).
Fields of papers citing papers by Ralf Egner
This network shows the impact of papers produced by Ralf Egner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ralf Egner. The network helps show where Ralf Egner may publish in the future.
Co-authors
The 18 scholars most cited alongside Ralf Egner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1994 | 491 | |
| 2 | 1998 | 160 | |
| 3 | 1998 | 132 | |
| 4 | 1995 | 119 | |
| 5 | 1996 | 109 | |
| 6 | 1993 | 73 | |
| 7 | 2000 | 69 | |
| 8 | 1995 | 17 | |
| 9 | 1997 | 8 | |
| 10 | 1995 | 3 | |
| 11 | 1997 | 2 |
About Ralf Egner
Ralf Egner is a scholar working on Oncology, Cell Biology, Molecular Biology, Infectious Diseases and Nutrition and Dietetics, having authored 11 papers that have together received 1.2k indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (5 papers), Cellular transport and secretion (4 papers), HIV/AIDS drug development and treatment (3 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Trace Elements in Health (2 papers), Advanced Proteomics Techniques and Applications (1 paper), Ubiquitin and proteasome pathways (1 paper) and Adenosine and Purinergic Signaling (1 paper). The work is most often cited by research in Cell Biology (502 citations), Epidemiology (562 citations), Physiology (73 citations), Molecular Biology (654 citations) and Oncology (197 citations). Ralf Egner has collaborated with scholars based in Austria, Germany and Switzerland. Frequent co-authors include Karl Kuchler, Michael Straub, Detlef Wolf, Michael Thumm, Martin Schlumpberger, Birgit Koch, Marten Veenhuis, Dieter H. Wolf, Richard K. Plemper and Rudy Pandjaitan. Their work appears in journals such as FEBS Letters, Journal of Biological Chemistry, Yeast, Molecular and Cellular Biology and Molecular Biology of the Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.