Quie Pg
Impact in
-
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms
- Immune Response and Inflammation
- Immunodeficiency and Autoimmune Disorders
- Microbiology top 10%
Papers in
-
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 4
- Immune Response and Inflammation 4
- Co-authors
- Good Ra (1 shared paper)John V. White (1 shared paper)Arne Forsgren (1 shared paper)Michael Af (1 shared paper)Williams Rc (1 shared paper)I. Ginsburg (1 shared paper)Meir Lahav (1 shared paper)Simmons Rl (1 shared paper)
- Journals
- Progress in clinical and biological research (1 paper)PubMed (20 papers)
- Partner nations
- United States
In The Last Decade
Quie Pg
20 papers receiving 387 citations
Peers
Comparison fields: 5 of 91
- Immunology 184
- Microbiology 40
- Molecular Medicine 29
- Infectious Diseases 100
- Immunology and Allergy 31
Countries citing papers authored by Quie Pg
This map shows the geographic impact of Quie Pg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Quie Pg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Quie Pg more than expected).
Fields of papers citing papers by Quie Pg
This network shows the impact of papers produced by Quie Pg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Quie Pg. The network helps show where Quie Pg may publish in the future.
Co-authors
The 10 scholars most cited alongside Quie Pg, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Fatal (chronic) granulomatous disease of childhood: a hereditary defect of leukocyte function. | 1968 | 96 |
| 2 | Comparison of phagocytic and chemiluminescence response of human polymorphonuclear neutrophils. | 1977 | 64 |
| 3 | Evaluation of a cytocentrifuge method for measuring neutrophil granulocyte chemotaxis. | 1975 | 62 |
| 4 | Severe staphylococcal disease associated with allergic manifestations, hyperimmunoglobulinemia E, and defective neutrophil chemotaxis. | 1976 | 55 |
| 5 | Evaluation of the Bauer-Kirby-Sherris-Turck single-disc diffusion method of antibiotic susceptibility testing. | 1969 | 36 |
| 6 | Microcolonies (G-variants) of Staphylococcus aureus. | 1969 | 34 |
| 7 | Studies of the alternate pathway in chelated serum. | 1975 | 31 |
| 8 | Acute disseminated and chronic mucocutaneous candidiasis. | 1971 | 29 |
| 9 | Molecular events during phagocytosis by human neutrophils. | 1977 | 19 |
| 10 | Studies of immune and normal opsonins during experimental staphylococcal infection in rabbits. | 1969 | 13 |
| 11 | Streptococcal and staphylococcal arthritis: can chronic arthritis in the human be caused by highly chemotactic degradation products generated from bacteria by leukocyte enzymes and by the deactivation of leukocytes by inflammatory exudates, polyelectrolytes, leukocyte hydrolases and by cell sensitizing agents derived from bacteria? | 1980 | 8 |
| 12 | The phagocytic system in host defense. | 1980 | 7 |
| 13 | D. pneumoniae. Increased resistance to oxacillin and cloxacillin. | 1968 | 7 |
| 14 | Disorders of phagocyte function: biochemical aspects. | 1977 | 4 |
| 15 | Potentiation of certain barbiturates by alpha-tocopherol phosphate. | 1954 | 2 |
| 16 | Phagocytes vs. bacteria, biological detente. | 1984 | 2 |
| 17 | The alternate complement pathway. A possible role in a patient with focal glomerular sclerosis. | 1981 | 2 |
| 18 | Serum opsonins for early diagnosis of disseminated candidiasis. | 1977 | 1 |
| 19 | Interaction of Staphylococcus aureus with human polymorphonuclear leukocytes. | 1973 | 1 |
| 20 | Antibiotic therapy and hospital-acquired infections. | 1969 | 1 |
About Quie Pg
Quie Pg is a scholar working on Immunology, Epidemiology, Molecular Biology, Pharmacology and Infectious Diseases, having authored 21 papers that have together received 475 indexed citations. Recurring topics across this work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers), Immune Response and Inflammation (4 papers), S100 Proteins and Annexins (2 papers), Antifungal resistance and susceptibility (2 papers), Cell Adhesion Molecules Research (2 papers), Blood disorders and treatments (2 papers), Streptococcal Infections and Treatments (1 paper) and Inhalation and Respiratory Drug Delivery (1 paper). The work is most often cited by research in Immunology (184 citations), Microbiology (40 citations), Molecular Medicine (29 citations), Infectious Diseases (100 citations) and Immunology and Allergy (31 citations). Quie Pg has collaborated with scholars based in United States. Frequent co-authors include Good Ra, John V. White, Arne Forsgren, Michael Af, Williams Rc, I. Ginsburg, Meir Lahav, Simmons Rl, Cuppage Fe and Joseph S. Solomkin. Their work appears in journals such as Progress in clinical and biological research and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.