Peter D. Feil

36 papers receiving 1.5k citations

Peers

Peter D. Feil
Comparison fields: 5 of 92
  • Genetics 1.2k
  • Reproductive Medicine 232
  • Toxicology 77
  • Endocrinology, Diabetes and Metabolism 303
  • Immunology 267
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Countries citing papers authored by Peter D. Feil

Since Specialization
Citations

This map shows the geographic impact of Peter D. Feil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter D. Feil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter D. Feil more than expected).

Fields of papers citing papers by Peter D. Feil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter D. Feil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter D. Feil. The network helps show where Peter D. Feil may publish in the future.

Co-authors

The 25 scholars most cited alongside Peter D. Feil, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Peter D. Feil Line = papers co-authored together Peter D. Feil links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1984373
2 1972214
3 1987126
4 1987121
5 1983119
6 1974116
7 1986107
8 198674
9 198852
10
Promotion by prolactin of the growth of human breast neoplasms cultured in vitro in the soft agar clonogenic assay.
198652
11
Autocrine stimulation by estradiol-regulated growth factors of rat hormone-responsive mammary cancer: interaction with the polyamine pathway.
198637
12
Progesterone receptor and regulation of progestin action in mammalian tissues.
197833
13 197726
14 197921
15 198721
16
Progesterone receptor structure and protease activity in primary human endometrial carcinoma.
198821
17 197619
18 198318
19 198917
20 198317

About Peter D. Feil

Peter D. Feil is a scholar working on Genetics, Molecular Biology, Reproductive Medicine, Immunology and Oncology, having authored 36 papers that have together received 1.7k indexed citations. Recurring topics across this work include Estrogen and related hormone effects (21 papers), Reproductive System and Pregnancy (5 papers), Ovarian function and disorders (3 papers), Monoclonal and Polyclonal Antibodies Research (3 papers), Hormonal and reproductive studies (2 papers), Plant-Derived Bioactive Compounds (2 papers), Cancer Diagnosis and Treatment (2 papers) and Breast Cancer Treatment Studies (2 papers). The work is most often cited by research in Genetics (1.2k citations), Reproductive Medicine (232 citations), Toxicology (77 citations), Endocrinology, Diabetes and Metabolism (303 citations) and Immunology (267 citations). Peter D. Feil has collaborated with scholars based in United States and Germany. Frequent co-authors include Richard J. Santen, Steven J. Santner, C. Wayne Bardin, P.G. Satyaswaroop, Christine L. Clarke, Bert W. O’Malley, Stanley R. Glasser, David O. Toft, Andrea Manni and Carol Wright. Their work appears in journals such as Endocrinology, The Journal of Clinical Endocrinology & Metabolism, Steroids, Cell Biochemistry and Function and Annals of the New York Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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