Pepijn Schellen

2.4k citations
12 papers · 669 · h-index 10

Impact in

Papers in

    • Extracellular vesicles in disease 2
    • Glioma Diagnosis and Treatment 3
    • Virus-based gene therapy research 2

Pepijn Schellen

12 papers receiving 649 citations

Peers

Pepijn Schellen
Comparison fields: 5 of 75
  • Animal Science and Zoology 259
  • Infectious Diseases 293
  • Cancer Research 136
  • Genetics 91
  • Genetics 177
Replace Karen J. Scott with:
Karen J. Scott United Kingdom
Qi Jin China
Kui Nie China
Rodrigo Haddad Brazil
Ulrike Dirmeier United States
Pei‐Yu Chu Taiwan
Rafael Contreras-Galindo United States
Christian Perez‐Shibayama Switzerland
J. Michael Stolley United States
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Citations per field
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Citations per year

Countries citing papers authored by Pepijn Schellen

Since Specialization
Citations

This map shows the geographic impact of Pepijn Schellen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pepijn Schellen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pepijn Schellen more than expected).

Fields of papers citing papers by Pepijn Schellen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pepijn Schellen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pepijn Schellen. The network helps show where Pepijn Schellen may publish in the future.

Co-authors

The 25 scholars most cited alongside Pepijn Schellen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Pepijn Schellen Line = papers co-authored together Pepijn Schellen links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 2005167
2 2015156
3 200881
4 200872
5 201265
6 201449
7 200525
8 201419
9 200516
10 201715
11 20153
12 20151

About Pepijn Schellen

Pepijn Schellen is a scholar working on Molecular Biology, Genetics, Pulmonary and Respiratory Medicine, Animal Science and Zoology and Infectious Diseases, having authored 12 papers that have together received 669 indexed citations. Recurring topics across this work include Animal Virus Infections Studies (3 papers), Glioma Diagnosis and Treatment (3 papers), SARS-CoV-2 and COVID-19 Research (2 papers), Lung Cancer Treatments and Mutations (2 papers), Extracellular vesicles in disease (2 papers), Virus-based gene therapy research (2 papers), Antibiotic Resistance in Bacteria (2 papers) and Cholangiocarcinoma and Gallbladder Cancer Studies (1 paper). The work is most often cited by research in Animal Science and Zoology (259 citations), Infectious Diseases (293 citations), Cancer Research (136 citations), Genetics (91 citations) and Genetics (177 citations). Pepijn Schellen has collaborated with scholars based in Netherlands, United States and Sweden. Frequent co-authors include Peter J. M. Rottier, Bert Jan Haijema, Kazuya Nakamura, Haukeline H. Volders, Thomas Würdinger, Dannis G. van Vuurden, Esther Hulleman, Harry Vennema, Gertjan J.L. Kaspers and David P. Noske. Their work appears in journals such as Journal of Virology, Oncotarget, Microbial Drug Resistance, Molecular Cancer Therapeutics and Journal of Clinical Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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