Pazit Ben-Av

1.1k citations
10 papers · 938 · h-index 10

Impact in

    • Inflammatory mediators and NSAID effects
    • Cancer, Hypoxia, and Metabolism
    • NF-κB Signaling Pathways

Papers in

    • Protein Kinase Regulation and GTPase Signaling 3
    • Metabolism, Diabetes, and Cancer 2
    • Angiogenesis and VEGF in Cancer 2
    • Inflammatory mediators and NSAID effects 4

Pazit Ben-Av

10 papers receiving 906 citations

Peers

Pazit Ben-Av
Comparison fields: 5 of 98
  • Pharmacology 316
  • Cancer Research 135
  • Biochemistry 58
  • Immunology and Allergy 43
  • Molecular Biology 447
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Citations per field
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Citations per year

Countries citing papers authored by Pazit Ben-Av

Since Specialization
Citations

This map shows the geographic impact of Pazit Ben-Av's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pazit Ben-Av with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pazit Ben-Av more than expected).

Fields of papers citing papers by Pazit Ben-Av

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pazit Ben-Av. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pazit Ben-Av. The network helps show where Pazit Ben-Av may publish in the future.

Co-authors

The 24 scholars most cited alongside Pazit Ben-Av, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Pazit Ben-Av Line = papers co-authored together Pazit Ben-Av links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 1995471
2
Non-small cell lung cancer cycloxygenase activity and proliferation are inhibited by non-steroidal antiinflammatory drugs.
199897
3 199371
4 199870
5 198961
6 200154
7 199340
8 198932
9 198822
10
Phospholipase D-mediated hydrolysis of phosphatidylcholine: role in cell signalling.
199320

About Pazit Ben-Av

Pazit Ben-Av is a scholar working on Molecular Biology, Pharmacology, Genetics, Surgery and Genetics, having authored 10 papers that have together received 938 indexed citations. Recurring topics across this work include Inflammatory mediators and NSAID effects (4 papers), Protein Kinase Regulation and GTPase Signaling (3 papers), Metabolism, Diabetes, and Cancer (2 papers), Angiogenesis and VEGF in Cancer (2 papers), Estrogen and related hormone effects (2 papers), Pancreatic function and diabetes (1 paper), Sperm and Testicular Function (1 paper) and Coagulation, Bradykinin, Polyphosphates, and Angioedema (1 paper). The work is most often cited by research in Pharmacology (316 citations), Cancer Research (135 citations), Biochemistry (58 citations), Immunology and Allergy (43 citations) and Molecular Biology (447 citations). Pazit Ben-Av has collaborated with scholars based in Israel and United States. Frequent co-authors include Timothy Hla, Ronald L. Wilder, Leslie J. Crofford, Mordechai Liscovitch, Etta Livneh, Catherine H. Liu, Hagit Eldar-Finkelman, Toyoaki Hida, Alfredo Martı́nez and Julius Leyton. Their work appears in journals such as FEBS Letters, Biochimica et Biophysica Acta (BBA) - Biomembranes, Lung Cancer, European Journal of Biochemistry and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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