Mostafa Ejtehadifar

425 citations
17 papers · 332 · h-index 9

Impact in

  • Genetics top 10%
    • Mesenchymal stem cell research
    • Cancer, Hypoxia, and Metabolism
    • MicroRNA in disease regulation

Papers in

Mostafa Ejtehadifar

16 papers receiving 324 citations

Peers

Mostafa Ejtehadifar
Comparison fields: 5 of 78
  • Genetics 134
  • Cancer Research 53
  • Biomaterials 34
  • Developmental Neuroscience 7
  • Endocrinology 9
Replace Shalini Vellasamy with:
Shalini Vellasamy Malaysia
Maryam Pashaiasl Iran
Young Jong Ko South Korea
Carlo Astarita United States
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Linglu Jia China
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Citations per year

Countries citing papers authored by Mostafa Ejtehadifar

Since Specialization
Citations

This map shows the geographic impact of Mostafa Ejtehadifar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mostafa Ejtehadifar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mostafa Ejtehadifar more than expected).

Fields of papers citing papers by Mostafa Ejtehadifar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mostafa Ejtehadifar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mostafa Ejtehadifar. The network helps show where Mostafa Ejtehadifar may publish in the future.

Co-authors

The 25 scholars most cited alongside Mostafa Ejtehadifar, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mostafa Ejtehadifar Line = papers co-authored together Mostafa Ejtehadifar links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 2015187
2 201737
3 201718
4 202117
5 201510
6 20169
7 20159
8
The Isolation and Detection of Acanthamoeba in Rural Water Sources of Arak, Iran
20148
9 20158
10 20177
11 20226
12 20195
13 20235
14 20144
15 20181
16 20171
17 20250

About Mostafa Ejtehadifar

Mostafa Ejtehadifar is a scholar working on Molecular Biology, Immunology, Genetics, Pathology and Forensic Medicine and Cancer Research, having authored 17 papers that have together received 332 indexed citations. Recurring topics across this work include Lymphoma Diagnosis and Treatment (3 papers), Mesenchymal stem cell research (3 papers), Liver physiology and pathology (2 papers), Pluripotent Stem Cells Research (2 papers), Cancer, Hypoxia, and Metabolism (2 papers), Ubiquitin and proteasome pathways (1 paper), Galectins and Cancer Biology (1 paper) and Insects and Parasite Interactions (1 paper). The work is most often cited by research in Genetics (134 citations), Cancer Research (53 citations), Biomaterials (34 citations), Developmental Neuroscience (7 citations) and Endocrinology (9 citations). Mostafa Ejtehadifar has collaborated with scholars based in Iran, Portugal and Denmark. Frequent co-authors include Karim Shamsasenjan, Parvin Akbarzadehlaleh, Nima Dehdilani, Ali Akbar Movassaghpour, Mahshid Saleh, Ali Ganji, Ghasem Mosayebi, Ali Ghazavi, Masoud Darabi and Nasser Hashemi Goradel. Their work appears in journals such as Advanced Pharmaceutical Bulletin, Microbial Pathogenesis, Nutritional Neuroscience, Cells and Clinical and Experimental Pharmacology and Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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