Mostafa Ejtehadifar
Impact in
- Genetics top 10%
- Mesenchymal stem cell research
-
- Cancer, Hypoxia, and Metabolism
- MicroRNA in disease regulation
Papers in
-
- Pluripotent Stem Cells Research 2
-
- Immunotherapy and Immune Responses 2
- Co-authors
- Karim Shamsasenjan (5 shared papers)Parvin Akbarzadehlaleh (3 shared papers)Nima Dehdilani (2 shared papers)Ali Akbar Movassaghpour (1 shared paper)Mahshid Saleh (1 shared paper)Ghasem Mosayebi (5 shared papers)Ali Ganji (4 shared papers)Ali Ghazavi (4 shared papers)
In The Last Decade
Mostafa Ejtehadifar
16 papers receiving 311 citations
Peers
Comparison fields: 5 of 76
- Genetics 138
- Cancer Research 56
- Biomaterials 37
- Developmental Neuroscience 9
- Endocrinology 9
Countries citing papers authored by Mostafa Ejtehadifar
This map shows the geographic impact of Mostafa Ejtehadifar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mostafa Ejtehadifar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mostafa Ejtehadifar more than expected).
Fields of papers citing papers by Mostafa Ejtehadifar
This network shows the impact of papers produced by Mostafa Ejtehadifar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mostafa Ejtehadifar. The network helps show where Mostafa Ejtehadifar may publish in the future.
Co-authors
The 25 scholars most cited alongside Mostafa Ejtehadifar, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 178 | |
| 2 | 2017 | 35 | |
| 3 | 2017 | 18 | |
| 4 | 2021 | 16 | |
| 5 | 2015 | 10 | |
| 6 | 2016 | 9 | |
| 7 | 2015 | 9 | |
| 8 | The Isolation and Detection of Acanthamoeba in Rural Water Sources of Arak, Iran | 2014 | 8 |
| 9 | 2017 | 7 | |
| 10 | 2015 | 7 | |
| 11 | 2022 | 6 | |
| 12 | 2023 | 5 | |
| 13 | 2019 | 5 | |
| 14 | 2014 | 4 | |
| 15 | 2017 | 1 | |
| 16 | 2018 | 1 | |
| 17 | 2025 | 0 |
About Mostafa Ejtehadifar
Mostafa Ejtehadifar is a scholar working on Molecular Biology, Immunology, Cancer Research, Genetics and Pathology and Forensic Medicine, having authored 17 papers that have together received 319 indexed citations. Recurring topics across this work include Mesenchymal stem cell research (3 papers), Lymphoma Diagnosis and Treatment (3 papers), Cancer, Hypoxia, and Metabolism (3 papers), Liver physiology and pathology (2 papers), Immunotherapy and Immune Responses (2 papers), Pluripotent Stem Cells Research (2 papers), Chemokine receptors and signaling (1 paper) and MicroRNA in disease regulation (1 paper). The work is most often cited by research in Genetics (138 citations), Cancer Research (56 citations), Biomaterials (37 citations), Developmental Neuroscience (9 citations) and Endocrinology (9 citations). Mostafa Ejtehadifar has collaborated with scholars based in Iran, Portugal and Denmark. Frequent co-authors include Karim Shamsasenjan, Parvin Akbarzadehlaleh, Nima Dehdilani, Ali Akbar Movassaghpour, Mahshid Saleh, Ghasem Mosayebi, Ali Ganji, Ali Ghazavi, Ana Sofía Carvalho and Rune Matthiesen. Their work appears in journals such as Advanced Pharmaceutical Bulletin, Microbial Pathogenesis, Cancers, Nutritional Neuroscience and Cells.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.