Miriam Eckstein
Impact in
- Sensory Systems top 1%
- Ion Channels and Receptors
- Immunology top 10%
- Immune Cell Function and Interaction
- T-cell and B-cell Immunology
- Immune cells in cancer
Papers in
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- Connexins and lens biology 6
- Ion channel regulation and function 2
-
- Ion Channels and Receptors 8
- Co-authors
- Martin Vaeth (11 shared papers)Stefan Feske (10 shared papers)Rodrigo S. Lacruz (12 shared papers)Jun J. Yang (4 shared papers)Meerim K. Nurbaeva (6 shared papers)Friederike Berberich‐Siebelt (2 shared papers)Stuart E. Turvey (2 shared papers)Scott B. Cameron (2 shared papers)
- Journals
- Nature Communications (3 papers)Immunity (2 papers)Cell Calcium (1 paper)Science Signaling (1 paper)Cell Metabolism (1 paper)
- Partner nations
- United StatesGermanyCanada
In The Last Decade
Miriam Eckstein
20 papers receiving 1.3k citations
Miriam Eckstein's Hit Papers
Peers
Comparison fields: 5 of 88
- Sensory Systems 323
- Immunology 375
- Physiology 55
- Rheumatology 147
- Biochemistry 55
Countries citing papers authored by Miriam Eckstein
This map shows the geographic impact of Miriam Eckstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miriam Eckstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miriam Eckstein more than expected).
Fields of papers citing papers by Miriam Eckstein
This network shows the impact of papers produced by Miriam Eckstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miriam Eckstein. The network helps show where Miriam Eckstein may publish in the future.
Co-authors
The 25 scholars most cited alongside Miriam Eckstein, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 203 | |
| 2 | 2017 | 170 | |
| 3 | Mitochondrial dysfunction promotes the transition of precursor to terminally exhausted T cells through HIF-1α-mediated glycolytic reprogramming Hit paper breakdown → | 2023 | 159 |
| 4 | The glucose transporter GLUT3 controls T helper 17 cell responses through glycolytic-epigenetic reprogramming Hit paper breakdown → | 2022 | 157 |
| 5 | 2016 | 93 | |
| 6 | 2016 | 70 | |
| 7 | 2017 | 66 | |
| 8 | 2017 | 49 | |
| 9 | 2019 | 48 | |
| 10 | 2019 | 45 | |
| 11 | 2015 | 42 | |
| 12 | 2016 | 38 | |
| 13 | 2013 | 35 | |
| 14 | 2015 | 27 | |
| 15 | 2023 | 22 | |
| 16 | 2018 | 22 | |
| 17 | 2018 | 20 | |
| 18 | 2018 | 18 | |
| 19 | 2021 | 7 | |
| 20 | 2009 | 3 |
About Miriam Eckstein
Miriam Eckstein is a scholar working on Molecular Biology, Sensory Systems, Immunology, Rheumatology and Cell Biology, having authored 20 papers that have together received 1.3k indexed citations. Recurring topics across this work include Ion Channels and Receptors (8 papers), Connexins and lens biology (6 papers), Bone and Dental Protein Studies (4 papers), Immune Cell Function and Interaction (4 papers), Aldose Reductase and Taurine (3 papers), Cancer, Hypoxia, and Metabolism (3 papers), T-cell and B-cell Immunology (2 papers) and Ion channel regulation and function (2 papers). The work is most often cited by research in Sensory Systems (323 citations), Immunology (375 citations), Physiology (55 citations), Rheumatology (147 citations) and Biochemistry (55 citations). Miriam Eckstein has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Martin Vaeth, Stefan Feske, Rodrigo S. Lacruz, Jun J. Yang, Meerim K. Nurbaeva, Friederike Berberich‐Siebelt, Stuart E. Turvey, Scott B. Cameron, Georg Gasteiger and Sophia M. Hochrein. Their work appears in journals such as Nature Communications, Immunity, Cell Calcium, Science Signaling and Cell Metabolism.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.