Michael V. Sofroniew

52.9k citations
208 papers · 41.4k · 24 hit papers · h-index 93

Impact in

Papers in

Michael V. Sofroniew

206 papers receiving 40.5k citations

Michael V. Sofroniew's Hit Papers

Astrocytes in human central nervous system diseases: a frontier for new therapies 2023 · 269 citations
2690+4+9Years since publication50010001.5k

Peers

Michael V. Sofroniew
Comparison fields: 5 of 169
  • Developmental Neuroscience 9.3k
  • Neurology 12.6k
  • Cellular and Molecular Neuroscience 14.7k
  • Biological Psychiatry 1.5k
  • Behavioral Neuroscience 1.5k
Replace Michael E. Greenberg with:
Michael E. Greenberg United States
Helmut Kettenmann Germany
Michal Schwartz Israel
Nave Klaus‐Armin Germany
Richard M. Ransohoff United States
Steven A. Goldman United States
Alexei Verkhratsky United Kingdom
David H. Rowitch United States
Olle Lindvall Sweden
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Michael V. Sofroniew relative to Michael E. Greenberg United States Michael E. Greenberg's profile →
Citations per field
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Citations per year

Countries citing papers authored by Michael V. Sofroniew

Since Specialization
Citations

This map shows the geographic impact of Michael V. Sofroniew's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael V. Sofroniew with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael V. Sofroniew more than expected).

Fields of papers citing papers by Michael V. Sofroniew

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael V. Sofroniew. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael V. Sofroniew. The network helps show where Michael V. Sofroniew may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael V. Sofroniew, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael V. Sofroniew Line = papers co-authored together Michael V. Sofroniew links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 208 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Astrocytes: biology and pathology
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20094128
2
Molecular dissection of reactive astrogliosis and glial scar formation
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20092151
3
Reactive astrocyte nomenclature, definitions, and future directions
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20211721
4
Astrocyte scar formation aids central nervous system axon regeneration
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20161468
5
Reactive Astrocytes Protect Tissue and Preserve Function after Spinal Cord Injury
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20041311
6
Reactive Gliosis and the Multicellular Response to CNS Damage and Disease
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20141151
7
Nerve Growth Factor Signaling, Neuroprotection, and Neural Repair
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20011109
8
Astrocyte barriers to neurotoxic inflammation
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2015989
9
Diversity of astrocyte functions and phenotypes in neural circuits
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2015940
10
Leukocyte Infiltration, Neuronal Degeneration, and Neurite Outgrowth after Ablation of Scar-Forming, Reactive Astrocytes in Adult Transgenic Mice
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1999933
11
Ablation of hippocampal neurogenesis impairs contextual fear conditioning and synaptic plasticity in the dentate gyrus
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2006854
12
GFAP-expressing progenitors are the principal source of constitutive neurogenesis in adult mouse forebrain
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2004809
13
STAT3 is a Critical Regulator of Astrogliosis and Scar Formation after Spinal Cord Injury
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2008798
14
Glial Scar Borders Are Formed by Newly Proliferated, Elongated Astrocytes That Interact to Corral Inflammatory and Fibrotic Cells via STAT3-Dependent Mechanisms after Spinal Cord Injury
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2013661
15 2005644
16
Astrocytes: a central element in neurological diseases
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2015625
17
Astrocyte roles in traumatic brain injury
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2015622
18 2007612
19
Astrocyte-derived VEGF-A drives blood-brain barrier disruption in CNS inflammatory disease
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2012602
20
Transformation of nonfunctional spinal circuits into functional states after the loss of brain input
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2009581

About Michael V. Sofroniew

Michael V. Sofroniew is a scholar working on Developmental Neuroscience, Cellular and Molecular Neuroscience, Neurology, Endocrine and Autonomic Systems and Behavioral Neuroscience, having authored 208 papers that have together received 41.4k indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (63 papers), Neuroinflammation and Neurodegeneration Mechanisms (56 papers), Nerve injury and regeneration (49 papers), Neuroscience and Neuropharmacology Research (36 papers), Neuroendocrine regulation and behavior (23 papers), Neuropeptides and Animal Physiology (17 papers), Neuroscience of respiration and sleep (15 papers) and Stress Responses and Cortisol (13 papers). The work is most often cited by research in Developmental Neuroscience (9.3k citations), Neurology (12.6k citations), Cellular and Molecular Neuroscience (14.7k citations), Biological Psychiatry (1.5k citations) and Behavioral Neuroscience (1.5k citations). Michael V. Sofroniew has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Harry V. Vinters, Joshua E. Burda, Baljit S. Khakh, Yan Ao, Mark A. Anderson, Tetsuya Imura, Adolf Weindl, Timothy Mark O’Shea, Ngan Doan and William Cary Mobley. Their work appears in journals such as Journal of Neuroscience, Neuroscience, Nature Neuroscience, Brain Research and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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