Michael Gale

46.9k citations
329 papers · 34.4k · 20 hit papers · h-index 95

Impact in

  • Hepatology top 0.05%
    • Hepatitis C virus research
  • Immunology top 0.02%
    • interferon and immune responses
    • Immune Response and Inflammation
    • Immune Cell Function and Interaction

Papers in

    • interferon and immune responses 172
    • Immune Response and Inflammation 38
    • Inflammasome and immune disorders 33

Michael Gale

321 papers receiving 33.8k citations

Michael Gale's Hit Papers

STING-induced regulatory B cells compromise NK function in cancer immunity 2022 · 235 citations
2350+7+14Years since publication50010001.5k

Peers

Michael Gale
Comparison fields: 5 of 165
  • Hepatology 7.2k
  • Immunology 17.8k
  • Infectious Diseases 8.2k
  • Virology 2.0k
  • Epidemiology 9.1k
Replace Yoshiharu Matsuura with:
Yoshiharu Matsuura Japan
Paul Klenerman United Kingdom
E. John Wherry United States
Francis V. Chisari United States
Glen N. Barber United States
Charles M. Rice United States
John Sidney United States
Michael B. A. Oldstone United States
Mary Carrington United States
Kunitada Shimotohno Japan
Michael Gale relative to Yoshiharu Matsuura Japan Yoshiharu Matsuura's profile →
Citations per field
00.5×3.3×
Yoshiharu Matsuura · 1×
Citations per year

Countries citing papers authored by Michael Gale

Since Specialization
Citations

This map shows the geographic impact of Michael Gale's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Gale with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Gale more than expected).

Fields of papers citing papers by Michael Gale

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Gale. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Gale. The network helps show where Michael Gale may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael Gale, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael Gale Line = papers co-authored together Michael Gale links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 329 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Immune Signaling by RIG-I-like Receptors
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20111509
2
Shared and Unique Functions of the DExD/H-Box Helicases RIG-I, MDA5, and LGP2 in Antiviral Innate Immunity
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20051392
3
Viruses and interferon: a fight for supremacy
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20021153
4
Immune evasion by hepatitis C virus NS3/4A protease-mediated cleavage of the Toll-like receptor 3 adaptor protein TRIF
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2005894
5
Distinct RIG-I and MDA5 Signaling by RNA Viruses in Innate Immunity
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2007870
6
Regulating Intracellular Antiviral Defense and Permissiveness to Hepatitis C Virus RNA Replication through a Cellular RNA Helicase, RIG-I
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2005738
7
2′-O methylation of the viral mRNA cap evades host restriction by IFIT family members
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2010700
8
Evidence That Hepatitis C Virus Resistance to Interferon Is Mediated through Repression of the PKR Protein Kinase by the Nonstructural 5A Protein
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1997697
9
Regulation of Interferon Regulatory Factor-3 by the Hepatitis C Virus Serine Protease
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2003644
10
Regulation of innate antiviral defenses through a shared repressor domain in RIG-I and LGP2
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2006615
11
Innate immunity induced by composition-dependent RIG-I recognition of hepatitis C virus RNA
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2008597
12
Control of PKR Protein Kinase by Hepatitis C Virus Nonstructural 5A Protein: Molecular Mechanisms of Kinase Regulation
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1998517
13
Small self-RNA generated by RNase L amplifies antiviral innate immunity
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2007509
14
Evasion of intracellular host defence by hepatitis C virus
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2005503
15
Inhibition of Retinoic Acid-Inducible Gene I-Mediated Induction of Beta Interferon by the NS1 Protein of Influenza A Virus
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2006500
16 2005467
17 2007445
18 2012430
19 2011416
20 2008396

About Michael Gale

Michael Gale is a scholar working on Immunology, Molecular Biology, Infectious Diseases, Epidemiology and Hepatology, having authored 329 papers that have together received 34.4k indexed citations. Recurring topics across this work include interferon and immune responses (172 papers), Hepatitis C virus research (73 papers), Mosquito-borne diseases and control (64 papers), Viral Infections and Vectors (59 papers), Immune Response and Inflammation (38 papers), Influenza Virus Research Studies (33 papers), HIV Research and Treatment (33 papers) and Inflammasome and immune disorders (33 papers). The work is most often cited by research in Hepatology (7.2k citations), Immunology (17.8k citations), Infectious Diseases (8.2k citations), Virology (2.0k citations) and Epidemiology (9.1k citations). Michael Gale has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Yueh–Ming Loo, Michael G. Katze, Eileen Foy, Michael Diamond, Stanley M. Lemon, Mehul S. Suthar, Kui Li, Yupeng He, Takashi Fujita and David M. Owen. Their work appears in journals such as Journal of Virology, PLoS Pathogens, The Journal of Immunology, Proceedings of the National Academy of Sciences and Journal of Interferon & Cytokine Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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