Michael De Lay
Impact in
- Cancer Research top 5%
- MicroRNA in disease regulation
- Cancer, Hypoxia, and Metabolism
- Cancer-related molecular mechanisms research
- Genetics top 5%
- Glioma Diagnosis and Treatment
- Chronic Lymphocytic Leukemia Research
Papers in
- Genetics 6
- Chronic Lymphocytic Leukemia Research 3
- Glioma Diagnosis and Treatment 3
- Co-authors
- Manish K. Aghi (8 shared papers)Arman Jahangiri (8 shared papers)Michal O. Nowicki (1 shared paper)James R. Van Brocklyn (1 shared paper)Michael C. Ostrowski (1 shared paper)Sean Lawler (1 shared paper)E. Antonio Chiocca (1 shared paper)Agnieszka Bronisz (1 shared paper)
- Journals
- Blood (3 papers)Neurosurgery (2 papers)Molecular Cell (1 paper)Clinical Cancer Research (1 paper)Autophagy (1 paper)
- Partner nations
- United StatesJapan
In The Last Decade
Michael De Lay
11 papers receiving 928 citations
Peers
Comparison fields: 5 of 58
- Cancer Research 372
- Genetics 223
- Immunology 162
- Oncology 188
- Molecular Biology 515
Countries citing papers authored by Michael De Lay
This map shows the geographic impact of Michael De Lay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael De Lay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael De Lay more than expected).
Fields of papers citing papers by Michael De Lay
This network shows the impact of papers produced by Michael De Lay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael De Lay. The network helps show where Michael De Lay may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael De Lay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 370 | |
| 2 | 2013 | 168 | |
| 3 | 2017 | 116 | |
| 4 | 2006 | 100 | |
| 5 | 2017 | 65 | |
| 6 | 2017 | 56 | |
| 7 | 2012 | 55 | |
| 8 | 2015 | 8 | |
| 9 | 2016 | 4 | |
| 10 | 2016 | 2 | |
| 11 | 2007 | 1 | |
| 12 | 2006 | 0 |
About Michael De Lay
Michael De Lay is a scholar working on Molecular Biology, Genetics, Immunology, Cancer Research and Pathology and Forensic Medicine, having authored 12 papers that have together received 945 indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (3 papers), Glioma Diagnosis and Treatment (3 papers), Cell Adhesion Molecules Research (2 papers), Macrophage Migration Inhibitory Factor (2 papers), MicroRNA in disease regulation (2 papers), Nuclear Receptors and Signaling (2 papers), Cancer, Hypoxia, and Metabolism (2 papers) and Cancer Mechanisms and Therapy (1 paper). The work is most often cited by research in Cancer Research (372 citations), Genetics (223 citations), Immunology (162 citations), Oncology (188 citations) and Molecular Biology (515 citations). Michael De Lay has collaborated with scholars based in United States and Japan. Frequent co-authors include Manish K. Aghi, Arman Jahangiri, Michal O. Nowicki, James R. Van Brocklyn, Michael C. Ostrowski, Sean Lawler, E. Antonio Chiocca, Agnieszka Bronisz, Gerard J. Nuovo and Jakub Godlewski. Their work appears in journals such as Blood, Neurosurgery, Molecular Cell, Clinical Cancer Research and Autophagy.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.