Michael Closser
Impact in
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- Neurogenesis and neuroplasticity mechanisms
Papers in
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- CRISPR and Genetic Engineering 5
- Pluripotent Stem Cells Research 4
- Genomics and Chromatin Dynamics 4
- RNA Research and Splicing 4
- Congenital heart defects research 2
- Genetics 2
- Neurogenetic and Muscular Disorders Research 2
- Co-authors
- Hynek Wichterle (10 shared papers)David K. Gifford (8 shared papers)Yuchun Guo (3 shared papers)Elizaveta Bashkirova (1 shared paper)Ho Sung Rhee (1 shared paper)Esteban O. Mazzoni (4 shared papers)Michelina Iacovino (2 shared papers)Tulsi Patel (2 shared papers)
- Journals
- Nature Methods (2 papers)Neuron (2 papers)Nature Communications (1 paper)Science Immunology (1 paper)Nature Neuroscience (1 paper)
- Partner nations
- United StatesSwitzerlandSingapore
In The Last Decade
Michael Closser
11 papers receiving 244 citations
Peers
Comparison fields: 5 of 40
- Developmental Neuroscience 23
- Aging 10
- Molecular Biology 213
- Genetics 18
- Cancer Research 21
Countries citing papers authored by Michael Closser
This map shows the geographic impact of Michael Closser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Closser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Closser more than expected).
Fields of papers citing papers by Michael Closser
This network shows the impact of papers produced by Michael Closser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Closser. The network helps show where Michael Closser may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael Closser, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 64 | |
| 2 | 2011 | 58 | |
| 3 | 2023 | 41 | |
| 4 | 2022 | 24 | |
| 5 | 2021 | 21 | |
| 6 | 2022 | 17 | |
| 7 | 2015 | 6 | |
| 8 | 2019 | 6 | |
| 9 | 2011 | 5 | |
| 10 | 2013 | 5 | |
| 11 | 2025 | 1 |
About Michael Closser
Michael Closser is a scholar working on Molecular Biology, Genetics, Plant Science, Developmental Neuroscience and Immunology, having authored 11 papers that have together received 248 indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (5 papers), Pluripotent Stem Cells Research (4 papers), Genomics and Chromatin Dynamics (4 papers), RNA Research and Splicing (4 papers), Neurogenetic and Muscular Disorders Research (2 papers), Chromosomal and Genetic Variations (2 papers), Congenital heart defects research (2 papers) and Neurogenesis and neuroplasticity mechanisms (1 paper). The work is most often cited by research in Developmental Neuroscience (23 citations), Aging (10 citations), Molecular Biology (213 citations), Genetics (18 citations) and Cancer Research (21 citations). Michael Closser has collaborated with scholars based in United States, Switzerland and Singapore. Frequent co-authors include Hynek Wichterle, David K. Gifford, Yuchun Guo, Elizaveta Bashkirova, Ho Sung Rhee, Esteban O. Mazzoni, Michelina Iacovino, Tulsi Patel, Michael Kyba and Shaun Mahony. Their work appears in journals such as Nature Methods, Neuron, Nature Communications, Science Immunology and Nature Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.