Michael C. Ewing

941 citations
13 papers · 755 · h-index 9

Impact in

    • Endoplasmic Reticulum Stress and Disease
    • Cellular transport and secretion
    • Protease and Inhibitor Mechanisms

Papers in

Michael C. Ewing

13 papers receiving 724 citations

Peers

Michael C. Ewing
Comparison fields: 5 of 72
  • Cell Biology 246
  • Cancer Research 159
  • Epidemiology 307
  • Physiology 44
  • Aging 16
Replace Mujun Zhao with:
Mujun Zhao China
Lea Mørch Harder Denmark
Christopher Cervantes United States
Sebastian Christopher Benjamin Bremer Germany
Peter Pyrko United States
Binayak Roy United States
John-Paul Upton United States
Qin Yu United States
Robert A. Saxton United States
Nicole C. McKnight United States
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Citations per field
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Citations per year

Countries citing papers authored by Michael C. Ewing

Since Specialization
Citations

This map shows the geographic impact of Michael C. Ewing's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael C. Ewing with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael C. Ewing more than expected).

Fields of papers citing papers by Michael C. Ewing

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael C. Ewing. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael C. Ewing. The network helps show where Michael C. Ewing may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael C. Ewing, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael C. Ewing Line = papers co-authored together Michael C. Ewing links everyone, so they are left out of the graph.

All Works

13 of 13 papers shown
#Work
1 2010515
2 200764
3 201439
4 201529
5 202226
6 201919
7 201116
8 201816
9 200513
10 20208
11 20214
12 20254
13 20252

About Michael C. Ewing

Michael C. Ewing is a scholar working on Cell Biology, Biotechnology, Aging, Molecular Biology and Epidemiology, having authored 13 papers that have together received 755 indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (4 papers), Autophagy in Disease and Therapy (3 papers), Bacillus and Francisella bacterial research (3 papers), Studies on Chitinases and Chitosanases (2 papers), Adipose Tissue and Metabolism (2 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers), Microbial Inactivation Methods (2 papers) and Insect Resistance and Genetics (1 paper). The work is most often cited by research in Cell Biology (246 citations), Cancer Research (159 citations), Epidemiology (307 citations), Physiology (44 citations) and Aging (16 citations). Michael C. Ewing has collaborated with scholars based in United States, Canada and Italy. Frequent co-authors include Tunda Hidvegi, Pamela D. Hale, Caroline S. Beckett, David H. Perlmutter, Amitava Mukherjee, Carolyn Kemp, Christine S Dippold, N.M. Maurice, George K. Michalopoulos and Simon C. Watkins. Their work appears in journals such as PLoS ONE, Journal of Biological Chemistry, Cell Death and Differentiation, Proceedings of the National Academy of Sciences and Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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