John-Paul Upton

2.0k citations
5 papers · 1.4k · 2 hit papers · h-index 5

Impact in

Papers in

    • Endoplasmic Reticulum Stress and Disease 5
    • RNA regulation and disease 3
    • Cell death mechanisms and regulation 1
    • CRISPR and Genetic Engineering 1

John-Paul Upton

5 papers receiving 1.4k citations

John-Paul Upton's Hit Papers

IRE1α Cleaves Select microRNAs During ER Stress to Derepress Translation of Proapoptotic Caspase-2 2012 · 532 citations
5320+5+11Years since publication200400600

Peers

John-Paul Upton
Comparison fields: 5 of 74
  • Cell Biology 1.1k
  • Aging 37
  • Epidemiology 660
  • Geriatrics and Gerontology 42
  • Molecular Biology 659
Replace Miho Oyadomari with:
Miho Oyadomari Japan
Haibo Sha United States
J. Liang United States
Aaron S. Mendez United States
Likun Wang China
Jeffrey J. Rodvold United States
Duc Thang Nguyên Canada
Arisa Higa Japan
Kwang‐Pyo Lee South Korea
Sean P. Ferris United States
John-Paul Upton relative to Miho Oyadomari Japan Miho Oyadomari's profile →
Citations per field
00.5×3.3×
Miho Oyadomari · 1×
Citations per year

Countries citing papers authored by John-Paul Upton

Since Specialization
Citations

This map shows the geographic impact of John-Paul Upton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John-Paul Upton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John-Paul Upton more than expected).

Fields of papers citing papers by John-Paul Upton

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John-Paul Upton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John-Paul Upton. The network helps show where John-Paul Upton may publish in the future.

Co-authors

The 19 scholars most cited alongside John-Paul Upton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John-Paul Upton Line = papers co-authored together John-Paul Upton links everyone, so they are left out of the graph.

All Works

5 of 5 papers shown

About John-Paul Upton

John-Paul Upton is a scholar working on Cell Biology, Molecular Biology, Surgery, Epidemiology and Infectious Diseases, having authored 5 papers that have together received 1.4k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (5 papers), RNA regulation and disease (3 papers), Pancreatic function and diabetes (2 papers), Autophagy in Disease and Therapy (2 papers), Cell death mechanisms and regulation (1 paper) and CRISPR and Genetic Engineering (1 paper). The work is most often cited by research in Cell Biology (1.1k citations), Aging (37 citations), Epidemiology (660 citations), Geriatrics and Gerontology (42 citations) and Molecular Biology (659 citations). John-Paul Upton has collaborated with scholars based in United States. Frequent co-authors include Feroz R. Papa, Dan Han, Scott A. Oakes, Andrew Hagen, Lieselotte Vande Walle, Bradley J. Backes, Alana G. Lerner, Weihong Xu, Andrei Goga and Lionel Lim. Their work appears in journals such as Cell, Science, Biochemical and Biophysical Research Communications and Molecular and Cellular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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