Michael B. Brenner

58.1k citations
238 papers · 36.0k · 15 hit papers · h-index 94

Impact in

  • Immunology top 0.01%
    • Immune Cell Function and Interaction
    • T-cell and B-cell Immunology
    • Immunotherapy and Immune Responses
    • Immune Response and Inflammation

Papers in

    • Immune Cell Function and Interaction 129
    • T-cell and B-cell Immunology 104
    • Immunotherapy and Immune Responses 51
    • Immune Response and Inflammation 22
    • Galectins and Cancer Biology 12

Michael B. Brenner

236 papers receiving 35.4k citations

Michael B. Brenner's Hit Papers

Fibroblasts as immune regulators in infection, inflammation and cancer 2021 · 390 citations
3900+13+26Years since publication10002.0k3.0k4.0k

Peers

Michael B. Brenner
Comparison fields: 5 of 173
  • Immunology 22.1k
  • Immunology and Allergy 1.7k
  • Oncology 4.4k
  • Infectious Diseases 2.7k
  • Epidemiology 4.1k
Replace Diane Mathis with:
Diane Mathis United States
Ronald N. Germain United States
Takashi Saito Japan
Hans D. Ochs United States
Ethan M. Shevach United States
William E. Paul United States
John W. Kappler United States
Sebastián Amigorena France
Alain Fischer France
Ko Okumura Japan
Michael B. Brenner relative to Diane Mathis United States Diane Mathis's profile →
Citations per field
00.5×1.5×
Diane Mathis · 1×
Citations per year

Countries citing papers authored by Michael B. Brenner

Since Specialization
Citations

This map shows the geographic impact of Michael B. Brenner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael B. Brenner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael B. Brenner more than expected).

Fields of papers citing papers by Michael B. Brenner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael B. Brenner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael B. Brenner. The network helps show where Michael B. Brenner may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael B. Brenner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael B. Brenner Line = papers co-authored together Michael B. Brenner links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 238 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Fast, sensitive and accurate integration of single-cell data with Harmony
Hit paper breakdown →
20194573
2
Adhesion between epithelial cells and T lymphocytes mediated by E-cadherin and the αEβ7 integrin
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1994947
3
Identification of a putative second T-cell receptor
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1986851
4
Natural and synthetic non-peptide antigens recognized by human γδ T cells
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1995849
5
Recognition of a lipid antigen by CD1-restricted αβ+ T cells
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1994833
6
CD1: Antigen Presentation and T Cell Function
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2004830
7
M. tuberculosis and M. leprae Translocate from the Phagolysosome to the Cytosol in Myeloid Cells
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2007741
8
Functionally Distinct Subsets of CD1d-restricted Natural Killer T Cells Revealed by CD1d Tetramer Staining
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2002628
9
CD1-Restricted T Cell Recognition of Microbial Lipoglycan Antigens
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1995627
10
Mast Cells: A Cellular Link Between Autoantibodies and Inflammatory Arthritis
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2002624
11
Analysis of T cell antigen receptor (TCR) expression by human peripheral blood CD4-8- alpha/beta T cells demonstrates preferential use of several V beta genes and an invariant TCR alpha chain.
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1993612
12
Arthritis Critically Dependent on Innate Immune System Players
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2002567
13
Lymphocytes bearing antigen-specific γδ T-cell receptors accumulate in human infectious disease lesions
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1989518
14 1992492
15 1989472
16 1989466
17
Metabolic reprogramming of natural killer cells in obesity limits antitumor responses
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2018464
18 1994460
19 1990449
20 1997428

About Michael B. Brenner

Michael B. Brenner is a scholar working on Immunology, Molecular Biology, Radiology, Nuclear Medicine and Imaging, Oncology and Epidemiology, having authored 238 papers that have together received 36.0k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (129 papers), T-cell and B-cell Immunology (104 papers), Immunotherapy and Immune Responses (51 papers), Monoclonal and Polyclonal Antibodies Research (41 papers), Cell Adhesion Molecules Research (24 papers), Immune Response and Inflammation (22 papers), CAR-T cell therapy research (16 papers) and Galectins and Cancer Biology (12 papers). The work is most often cited by research in Immunology (22.1k citations), Immunology and Allergy (1.7k citations), Oncology (4.4k citations), Infectious Diseases (2.7k citations) and Epidemiology (4.1k citations). Michael B. Brenner has collaborated with scholars based in United States, United Kingdom and Netherlands. Frequent co-authors include Steven A. Porcelli, Craig T. Morita, Manfred Brigl, Kevin Wei, Soumya Raychaudhuri, Kamil Slowikowski, Fan Zhang, Ilya Korsunsky, Yuriy Baglaenko and Po‐Ru Loh. Their work appears in journals such as The Journal of Experimental Medicine, Proceedings of the National Academy of Sciences, The Journal of Immunology, Nature and Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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