Mathew Tata

603 citations
9 papers · 379 · h-index 8

Impact in

    • Neurogenesis and neuroplasticity mechanisms
    • Neuroinflammation and Neurodegeneration Mechanisms
    • Barrier Structure and Function Studies

Papers in

Mathew Tata

8 papers receiving 375 citations

Peers

Mathew Tata
Comparison fields: 5 of 62
  • Developmental Neuroscience 71
  • Neurology 52
  • Cellular and Molecular Neuroscience 92
  • Neurology 43
  • Physiology 68
Replace Alexandra I. Rosa with:
Alexandra I. Rosa Portugal
Ilaria Cervellini United Kingdom
Tae‐Ryong Riew South Korea
R.L. Zhang United States
Frisca Frisca Indonesia
Ganlan Bian China
Kazuhiko Namikawa Japan
Óscar G. Vidaurre Spain
Chuanxi Tang China
Sina K. Stumpf Germany
Mathew Tata relative to Alexandra I. Rosa Portugal Alexandra I. Rosa's profile →
Citations per field
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Alexandra I. Rosa · 1×
Citations per year

Countries citing papers authored by Mathew Tata

Since Specialization
Citations

This map shows the geographic impact of Mathew Tata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mathew Tata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mathew Tata more than expected).

Fields of papers citing papers by Mathew Tata

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mathew Tata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mathew Tata. The network helps show where Mathew Tata may publish in the future.

Co-authors

The 25 scholars most cited alongside Mathew Tata, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mathew Tata Line = papers co-authored together Mathew Tata links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 2015104
2 201365
3 199964
4 201839
5 201639
6 201827
7 201821
8 202520
9 20180

About Mathew Tata

Mathew Tata is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Developmental Neuroscience, Pharmacology and Cell Biology, having authored 9 papers that have together received 379 indexed citations. Recurring topics across this work include Axon Guidance and Neuronal Signaling (6 papers), Neurogenesis and neuroplasticity mechanisms (5 papers), Angiogenesis and VEGF in Cancer (3 papers), Epigenetics and DNA Methylation (1 paper), Genetic and Kidney Cyst Diseases (1 paper), Single-cell and spatial transcriptomics (1 paper), Myofascial pain diagnosis and treatment (1 paper) and Zebrafish Biomedical Research Applications (1 paper). The work is most often cited by research in Developmental Neuroscience (71 citations), Neurology (52 citations), Cellular and Molecular Neuroscience (92 citations), Neurology (43 citations) and Physiology (68 citations). Mathew Tata has collaborated with scholars based in United Kingdom, Mexico and Australia. Frequent co-authors include Christiana Ruhrberg, Alessandro Fantin, N Bons, D Albe‐Fessard, Aman Dalal, Ivan Wall, Joaquim Miguel Vieira, Nicoletta Kessaris, Sarah Leigh Nicholson and Thomas S. Jacques. Their work appears in journals such as Neuroscience, Mechanisms of Development, Acta Neuropathologica, Development and Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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