Mark Halliday

2.3k citations
14 papers · 1.9k · h-index 12

Impact in

Papers in

    • Endoplasmic Reticulum Stress and Disease 7
    • Prion Diseases and Protein Misfolding 5
    • Mitochondrial Function and Pathology 1

Mark Halliday

14 papers receiving 1.9k citations

Peers

Mark Halliday
Comparison fields: 5 of 71
  • Cell Biology 699
  • Aging 48
  • Neurology 192
  • Physiology 318
  • Molecular Biology 656
Replace Helois E. Radford with:
Helois E. Radford United Kingdom
Nicholas Verity United Kingdom
Jef Swerts Belgium
Heather Smith United States
Jeff W. Barclay United Kingdom
Eva Teuling Netherlands
Craig Meyers United States
Philipp A. Jaeger United States
Lukas Jakob Neukomm Switzerland
Ravindran Kumaran United States
Mark Halliday relative to Helois E. Radford United Kingdom Helois E. Radford's profile →
Citations per field
00.5×1.5×
Helois E. Radford · 1×
Citations per year

Countries citing papers authored by Mark Halliday

Since Specialization
Citations

This map shows the geographic impact of Mark Halliday's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Halliday with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Halliday more than expected).

Fields of papers citing papers by Mark Halliday

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Halliday. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Halliday. The network helps show where Mark Halliday may publish in the future.

Co-authors

The 25 scholars most cited alongside Mark Halliday, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark Halliday Line = papers co-authored together Mark Halliday links everyone, so they are left out of the graph.

All Works

14 of 14 papers shown
#Work
1 2012517
2 2013487
3 2015244
4 2017242
5 2013103
6 201882
7 201470
8 201752
9 201442
10 201721
11 202019
12 202315
13 20194
14 20232

About Mark Halliday

Mark Halliday is a scholar working on Cell Biology, Molecular Biology, Physiology, Computational Mechanics and Genetics, having authored 14 papers that have together received 1.9k indexed citations. Recurring topics across this work include Endoplasmic Reticulum Stress and Disease (7 papers), Prion Diseases and Protein Misfolding (5 papers), Alzheimer's disease research and treatments (2 papers), Down syndrome and intellectual disability research (1 paper), Genetics and Neurodevelopmental Disorders (1 paper), Mitochondrial Function and Pathology (1 paper) and Ion-surface interactions and analysis (1 paper). The work is most often cited by research in Cell Biology (699 citations), Aging (48 citations), Neurology (192 citations), Physiology (318 citations) and Molecular Biology (656 citations). Mark Halliday has collaborated with scholars based in United Kingdom, United States and Australia. Frequent co-authors include Giovanna R. Mallucci, Helois E. Radford, Nicholas Verity, Julie A. Moreno, David A. Barrett, Anne E. Willis, Colin Molloy, Catharine A. Ortori, Martin Bushell and Peter M. Fischer. Their work appears in journals such as Brain, Science Translational Medicine, Neuropharmacology, Journal of Neurochemistry and Acta Neuropathologica.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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