Mark Halliday

2.3k citations
14 papers · 1.8k · 2 hit papers · h-index 11

Impact in

Papers in

    • Prion Diseases and Protein Misfolding 10
    • RNA regulation and disease 2
    • Endoplasmic Reticulum Stress and Disease 7

Mark Halliday

14 papers receiving 1.7k citations

Mark Halliday's Hit Papers

Oral Treatment Targeting the Unfolded Protein Response Prevents Neurodegeneration and Clinical Disease in Prion-Infected Mice 2013 · 453 citations
4530+4+9Years since publication100200300400

Peers

Mark Halliday
Comparison fields: 5 of 83
  • Cell Biology 859
  • Aging 56
  • Neurology 238
  • Biological Psychiatry 62
  • Physiology 429
Replace Helois Radford with:
Helois Radford United Kingdom
Nicholas Verity United Kingdom
Elena Ziviani Italy
Colin Molloy United Kingdom
Riccardo Cristofani Italy
Álvaro Sánchez-Martínez United Kingdom
Véronique Schaeffer France
Salvatore J. Cherra United States
Samantha H. Y. Loh United Kingdom
Ramya Narasimhan United States
Mark Halliday relative to Helois Radford United Kingdom Helois Radford's profile →
Citations per field
00.5×1.5×
Helois Radford · 1×
Citations per year

Countries citing papers authored by Mark Halliday

Since Specialization
Citations

This map shows the geographic impact of Mark Halliday's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark Halliday with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark Halliday more than expected).

Fields of papers citing papers by Mark Halliday

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark Halliday. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark Halliday. The network helps show where Mark Halliday may publish in the future.

Co-authors

The 25 scholars most cited alongside Mark Halliday, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark Halliday Line = papers co-authored together Mark Halliday links everyone, so they are left out of the graph.

All Works

14 of 14 papers shown
#Work
1
Sustained translational repression by eIF2α-P mediates prion neurodegeneration
Hit paper breakdown →
2012489
2
Oral Treatment Targeting the Unfolded Protein Response Prevents Neurodegeneration and Clinical Disease in Prion-Infected Mice
Hit paper breakdown →
2013453
3 2015222
4 2017213
5 201398
6 201877
7 201464
8 201749
9 201442
10 201717
11 202013
12 20239
13 20108
14 20231

About Mark Halliday

Mark Halliday is a scholar working on Molecular Biology, Cell Biology, Physiology, Neurology and Cellular and Molecular Neuroscience, having authored 14 papers that have together received 1.8k indexed citations. Recurring topics across this work include Prion Diseases and Protein Misfolding (10 papers), Endoplasmic Reticulum Stress and Disease (7 papers), Alzheimer's disease research and treatments (4 papers), Neurological diseases and metabolism (3 papers), RNA regulation and disease (2 papers), Ion-surface interactions and analysis (1 paper), Trace Elements in Health (1 paper) and Histiocytic Disorders and Treatments (1 paper). The work is most often cited by research in Cell Biology (859 citations), Aging (56 citations), Neurology (238 citations), Biological Psychiatry (62 citations) and Physiology (429 citations). Mark Halliday has collaborated with scholars based in United Kingdom, Australia and Switzerland. Frequent co-authors include Giovanna R. Mallucci, Helois Radford, Nicholas Verity, Julie A. Moreno, David A. Barrett, Anne E. Willis, Colin Molloy, Catharine A. Ortori, Martin Bushell and Jeffrey M. Axten. Their work appears in journals such as Brain, Science Translational Medicine, Analytical Chemistry, Neuropharmacology and Cell Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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