M Comley

909 citations
12 papers · 763 · h-index 10

Impact in

Papers in

    • Glycosylation and Glycoproteins Research 2
    • PI3K/AKT/mTOR signaling in cancer 2
    • Angiogenesis and VEGF in Cancer 1
    • Vascular Tumors and Angiosarcomas 2

M Comley

12 papers receiving 743 citations

Peers

M Comley
Comparison fields: 5 of 58
  • Cancer Research 172
  • Oncology 230
  • Molecular Biology 515
  • Genetics 186
  • Toxicology 21
Replace CL Arteaga with:
CL Arteaga United States
М. В. Немцова Russia
Marie Dance France
Maozhen Tian United States
Jeanie Wu Singapore
María D. Corte‐Torres Spain
Silvia Tórtola Spain
Carlo Gatti Italy
Arezoo Astanehe Canada
Mélanie Mestdagt Belgium
M Comley relative to CL Arteaga United States CL Arteaga's profile →
Citations per field
00.5×4.2×
CL Arteaga · 1×
Citations per year

Countries citing papers authored by M Comley

Since Specialization
Citations

This map shows the geographic impact of M Comley's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Comley with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Comley more than expected).

Fields of papers citing papers by M Comley

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Comley. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Comley. The network helps show where M Comley may publish in the future.

Co-authors

The 25 scholars most cited alongside M Comley, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with M Comley Line = papers co-authored together M Comley links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 1996192
2 1997147
3 1996138
4 1997119
5 199754
6 199751
7 199715
8 199515
9
The distribution of the deleted in colon cancer (DCC) protein in human tissues.
199515
10 19979
11 19967
12 19971

About M Comley

M Comley is a scholar working on Molecular Biology, Oncology, Cancer Research, Pathology and Forensic Medicine and Genetics, having authored 12 papers that have together received 763 indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (3 papers), Glycosylation and Glycoproteins Research (2 papers), Vascular Tumors and Angiosarcomas (2 papers), PI3K/AKT/mTOR signaling in cancer (2 papers), Fetal and Pediatric Neurological Disorders (1 paper), Angiogenesis and VEGF in Cancer (1 paper), Bioactive Compounds and Antitumor Agents (1 paper) and Genetic and Kidney Cyst Diseases (1 paper). The work is most often cited by research in Cancer Research (172 citations), Oncology (230 citations), Molecular Biology (515 citations), Genetics (186 citations) and Toxicology (21 citations). M Comley has collaborated with scholars based in United Kingdom, Greece and South Africa. Frequent co-authors include Helen Turley, Adrian L. Harris, K C Gatter, Runjan Chetty, Kevin C. Gatter, Simon Biddolph, Stephen B. Fox, Alexandra Giatromanolaki, Christopher J. Ward and Peter J. Ratcliffe. Their work appears in journals such as British Journal of Cancer, The Journal of Pathology, Journal of Clinical Pathology, Annals of Oncology and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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