Luke E. Formosa
Impact in
- Molecular Biology top 5%
- Mitochondrial Function and Pathology
- Clinical Biochemistry top 5%
- Metabolism and Genetic Disorders
Papers in
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- Mitochondrial Function and Pathology 26
- PI3K/AKT/mTOR signaling in cancer 1
- Ion channel regulation and function 1
- Metabolism, Diabetes, and Cancer 1
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- Metabolism and Genetic Disorders 2
- Co-authors
- Michael T. Ryan (20 shared papers)David A. Stroud (12 shared papers)Marris G. Dibley (3 shared papers)Boris Reljić (6 shared papers)David R. Thorburn (6 shared papers)Tegan L. Stait (4 shared papers)Ann E. Frazier (3 shared papers)Traude Helene Beilharz (2 shared papers)
- Journals
- Nature Communications (3 papers)Proceedings of the National Academy of Sciences (3 papers)Human Mutation (2 papers)Neurobiology of Disease (1 paper)Biochimica et Biophysica Acta (BBA) - Bioenergetics (1 paper)
- Partner nations
- AustraliaUnited StatesUnited Kingdom
In The Last Decade
Luke E. Formosa
33 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 88
- Molecular Biology 1.1k
- Clinical Biochemistry 83
- Aging 16
- Cancer Research 57
- Genetics 110
Countries citing papers authored by Luke E. Formosa
This map shows the geographic impact of Luke E. Formosa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luke E. Formosa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luke E. Formosa more than expected).
Fields of papers citing papers by Luke E. Formosa
This network shows the impact of papers produced by Luke E. Formosa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luke E. Formosa. The network helps show where Luke E. Formosa may publish in the future.
Co-authors
The 25 scholars most cited alongside Luke E. Formosa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 451 | |
| 2 | 2017 | 166 | |
| 3 | 2020 | 86 | |
| 4 | 2017 | 80 | |
| 5 | 2019 | 75 | |
| 6 | 2012 | 73 | |
| 7 | 2018 | 66 | |
| 8 | 2015 | 63 | |
| 9 | 2016 | 55 | |
| 10 | 2019 | 55 | |
| 11 | 2022 | 55 | |
| 12 | 2020 | 52 | |
| 13 | 2018 | 47 | |
| 14 | 2020 | 47 | |
| 15 | 2020 | 27 | |
| 16 | 2021 | 22 | |
| 17 | 2022 | 22 | |
| 18 | 2022 | 21 | |
| 19 | 2016 | 20 | |
| 20 | 2021 | 18 |
About Luke E. Formosa
Luke E. Formosa is a scholar working on Molecular Biology, Clinical Biochemistry, Genetics, Nephrology and Cell Biology, having authored 33 papers that have together received 1.6k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (26 papers), Genomics and Rare Diseases (2 papers), Metabolism and Genetic Disorders (2 papers), PI3K/AKT/mTOR signaling in cancer (1 paper), Ion channel regulation and function (1 paper), Acute Kidney Injury Research (1 paper), Metabolism, Diabetes, and Cancer (1 paper) and Pancreatic function and diabetes (1 paper). The work is most often cited by research in Molecular Biology (1.1k citations), Clinical Biochemistry (83 citations), Aging (16 citations), Cancer Research (57 citations) and Genetics (110 citations). Luke E. Formosa has collaborated with scholars based in Australia, United States and United Kingdom. Frequent co-authors include Michael T. Ryan, David A. Stroud, Marris G. Dibley, Boris Reljić, David R. Thorburn, Tegan L. Stait, Ann E. Frazier, Traude Helene Beilharz, Elliot E. Surgenor and Laura Danielle Osellame. Their work appears in journals such as Nature Communications, Proceedings of the National Academy of Sciences, Human Mutation, Neurobiology of Disease and Biochimica et Biophysica Acta (BBA) - Bioenergetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.