Lorena Cussó

37 papers receiving 957 citations

Lorena Cussó's Hit Papers

T cells with dysfunctional mitochondria induce multimorbidity and premature senescence 2020 · 397 citations
3970+2+4Years since publication100200300

Peers

Lorena Cussó
Comparison fields: 5 of 114
  • Aging 36
  • Biological Psychiatry 30
  • Immunology 259
  • Neurology 59
  • Physiology 162
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Chunmei Piao China
Yumin Oh United States
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C Molina United Kingdom
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Citations per year

Countries citing papers authored by Lorena Cussó

Since Specialization
Citations

This map shows the geographic impact of Lorena Cussó's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lorena Cussó with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lorena Cussó more than expected).

Fields of papers citing papers by Lorena Cussó

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lorena Cussó. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lorena Cussó. The network helps show where Lorena Cussó may publish in the future.

Co-authors

The 25 scholars most cited alongside Lorena Cussó, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Lorena Cussó Line = papers co-authored together Lorena Cussó links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 38 papers — load more, or switch the sort, to bring in the rest.

#Work
1
T cells with dysfunctional mitochondria induce multimorbidity and premature senescence
Hit paper breakdown →
2020397
2 201473
3 202171
4 201770
5 201856
6 201949
7 201328
8 201922
9 202219
10 201417
11 201317
12 202114
13 202413
14 201413
15 202012
16 202212
17 20229
18 20199
19 20186
20 20196

About Lorena Cussó

Lorena Cussó is a scholar working on Radiology, Nuclear Medicine and Imaging, Molecular Biology, Biomedical Engineering, Surgery and Infectious Diseases, having authored 38 papers that have together received 965 indexed citations. Recurring topics across this work include Medical Imaging Techniques and Applications (6 papers), Advanced MRI Techniques and Applications (6 papers), Photoacoustic and Ultrasonic Imaging (3 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers), Orthopedic Infections and Treatments (3 papers), Infective Endocarditis Diagnosis and Management (3 papers), Clostridium difficile and Clostridium perfringens research (2 papers) and Mesenchymal stem cell research (2 papers). The work is most often cited by research in Aging (36 citations), Biological Psychiatry (30 citations), Immunology (259 citations), Neurology (59 citations) and Physiology (162 citations). Lorena Cussó has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Manuel Desco, Borja Ibáñez, Pablo J. Fernández-Marcos, Bruno Hernáez, Jorge Oller, Arántzazu Alfranca, Gabriela Desdín-Micó, Marı́a Mittelbrunn, Antonio Alcamı́ and Marı́a N. Navarro. Their work appears in journals such as Molecular Imaging and Biology, PLoS ONE, ChemPhotoChem, International Journal of Molecular Sciences and Biology Direct.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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