Leonardo Bee
Impact in
- Aging top 2%
- Genetics, Aging, and Longevity in Model Organisms
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- Circadian rhythm and melatonin
Papers in
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- DNA Repair Mechanisms 3
- CRISPR and Genetic Engineering 2
- Mitochondrial Function and Pathology 2
- RNA modifications and cancer 1
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- Circadian rhythm and melatonin 2
- Co-authors
- Paolo Sassone‐Corsi (1 shared paper)Guiomar Solanas (1 shared paper)Aikaterini Symeonidi (1 shared paper)Mark S. Schmidt (1 shared paper)Charles Brenner (1 shared paper)Salvador Aznar Benitah (1 shared paper)Selma Masri (1 shared paper)Shogo Sato (1 shared paper)
- Journals
- Nucleic Acids Research (1 paper)Proceedings of the National Academy of Sciences (1 paper)EMBO Molecular Medicine (1 paper)Cell Biology and Toxicology (1 paper)Cell (1 paper)
- Partner nations
- ItalyUnited StatesUnited Kingdom
In The Last Decade
Leonardo Bee
6 papers receiving 474 citations
Peers
Comparison fields: 5 of 62
- Aging 102
- Endocrine and Autonomic Systems 196
- Physiology 154
- Biological Psychiatry 12
- Geriatrics and Gerontology 13
Countries citing papers authored by Leonardo Bee
This map shows the geographic impact of Leonardo Bee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonardo Bee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonardo Bee more than expected).
Fields of papers citing papers by Leonardo Bee
This network shows the impact of papers produced by Leonardo Bee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonardo Bee. The network helps show where Leonardo Bee may publish in the future.
Co-authors
The 25 scholars most cited alongside Leonardo Bee, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 273 | |
| 2 | 2012 | 97 | |
| 3 | 2013 | 60 | |
| 4 | 2015 | 25 | |
| 5 | 2015 | 20 | |
| 6 | 2017 | 7 |
About Leonardo Bee
Leonardo Bee is a scholar working on Molecular Biology, Endocrine and Autonomic Systems, Oncology, Physiology and Cancer Research, having authored 6 papers that have together received 482 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (3 papers), CRISPR and Genetic Engineering (2 papers), Circadian rhythm and melatonin (2 papers), Mitochondrial Function and Pathology (2 papers), Carcinogens and Genotoxicity Assessment (2 papers), Cancer-related Molecular Pathways (1 paper), Electromagnetic Fields and Biological Effects (1 paper) and RNA modifications and cancer (1 paper). The work is most often cited by research in Aging (102 citations), Endocrine and Autonomic Systems (196 citations), Physiology (154 citations), Biological Psychiatry (12 citations) and Geriatrics and Gerontology (13 citations). Leonardo Bee has collaborated with scholars based in Italy, United States and United Kingdom. Frequent co-authors include Paolo Sassone‐Corsi, Guiomar Solanas, Aikaterini Symeonidi, Mark S. Schmidt, Charles Brenner, Salvador Aznar Benitah, Selma Masri, Shogo Sato, Paola Ferraro and Giovanna Pontarin. Their work appears in journals such as Nucleic Acids Research, Proceedings of the National Academy of Sciences, EMBO Molecular Medicine, Cell Biology and Toxicology and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.