L. Kasch

787 citations
7 papers · 666 · h-index 5

Impact in

Papers in

    • Cancer-related gene regulation 3
    • Ubiquitin and proteasome pathways 1
    • Connexins and lens biology 1
    • Receptor Mechanisms and Signaling 1
    • Hemophilia Treatment and Research 3
    • Hemostasis and retained surgical items 1

L. Kasch

7 papers receiving 637 citations

Peers

L. Kasch
Comparison fields: 5 of 61
  • Hematology 192
  • Cellular and Molecular Neuroscience 325
  • Molecular Biology 465
  • Biological Psychiatry 13
  • Genetics 39
Replace Brandon S. Willis with:
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Elisa Bofill-Cardona Austria
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Yukiko Nasu‐Nishimura Japan
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Citations per field
00.5×8.7×
Brandon S. Willis · 1×
Citations per year

Countries citing papers authored by L. Kasch

Since Specialization
Citations

This map shows the geographic impact of L. Kasch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L. Kasch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L. Kasch more than expected).

Fields of papers citing papers by L. Kasch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by L. Kasch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L. Kasch. The network helps show where L. Kasch may publish in the future.

Co-authors

The 25 scholars most cited alongside L. Kasch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with L. Kasch Line = papers co-authored together L. Kasch links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 1991378
2 1991127
3 1991108
4
Analysis of DNA polymorphism haplotypes linked to the cystic fibrosis locus in North American black and Caucasian families supports the existence of multiple mutations of the cystic fibrosis gene.
198944
5 20025
6 20232
7
Prenatal testing for Huntington disease.
19902

About L. Kasch

L. Kasch is a scholar working on Molecular Biology, Hematology, Cellular and Molecular Neuroscience, Pediatrics, Perinatology and Child Health and Endocrine and Autonomic Systems, having authored 7 papers that have together received 666 indexed citations. Recurring topics across this work include Hemophilia Treatment and Research (3 papers), Cancer-related gene regulation (3 papers), Gestational Diabetes Research and Management (1 paper), Ubiquitin and proteasome pathways (1 paper), Connexins and lens biology (1 paper), Neurological diseases and metabolism (1 paper), Receptor Mechanisms and Signaling (1 paper) and Hemostasis and retained surgical items (1 paper). The work is most often cited by research in Hematology (192 citations), Cellular and Molecular Neuroscience (325 citations), Molecular Biology (465 citations), Biological Psychiatry (13 citations) and Genetics (39 citations). L. Kasch has collaborated with scholars based in United States, Germany and Türkiye. Frequent co-authors include Stylianos E. Antonarakis, Garry R. Cutting, Lili Lu, Shoichi Shimada, Bruce F. O’Hara, William B. Guggino, Chahrzad Montrose‐Rafizadeh, David M. Donovan, George R. Uhl and Miyoko Higuchi. Their work appears in journals such as Proceedings of the National Academy of Sciences, Cytogenetic and Genome Research, Molecular Metabolism and PubMed.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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