Kerstin Willander

496 citations
10 papers · 364 · h-index 7

Impact in

Papers in

    • Chronic Lymphocytic Leukemia Research 5
    • Acute Myeloid Leukemia Research 5

Kerstin Willander

10 papers receiving 360 citations

Peers

Kerstin Willander
Comparison fields: 5 of 40
  • Genetics 237
  • Pathology and Forensic Medicine 155
  • Immunology 140
  • Hematology 74
  • Radiology, Nuclear Medicine and Imaging 50
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Citations per year

Countries citing papers authored by Kerstin Willander

Since Specialization
Citations

This map shows the geographic impact of Kerstin Willander's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kerstin Willander with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kerstin Willander more than expected).

Fields of papers citing papers by Kerstin Willander

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kerstin Willander. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kerstin Willander. The network helps show where Kerstin Willander may publish in the future.

Co-authors

The 25 scholars most cited alongside Kerstin Willander, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kerstin Willander Line = papers co-authored together Kerstin Willander links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 2008199
2 201546
3 201432
4 201329
5
Patients with chronic lymphocytic leukemia with mutated VH genes presenting with Binet stage B or C form a subgroup with a poor outcome.
200522
6 201416
7 201013
8
Production of transforming growth factor alpha by human leukemia cells (HL-60 and U-937) during monocytic differentiation.
19955
9 20151
10
VH gene mutation analysis distinguishes a subgroup of chronic lymphocytic leukemia with low-mutated VH genes and inferior outcome.
20041

About Kerstin Willander

Kerstin Willander is a scholar working on Genetics, Hematology, Molecular Biology, Oncology and Immunology, having authored 10 papers that have together received 364 indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (5 papers), Chronic Lymphocytic Leukemia Research (5 papers), Cancer Genomics and Diagnostics (2 papers), Cancer-related Molecular Pathways (2 papers), Glycosylation and Glycoproteins Research (2 papers), Lymphoma Diagnosis and Treatment (1 paper), Cytokine Signaling Pathways and Interactions (1 paper) and Galectins and Cancer Biology (1 paper). The work is most often cited by research in Genetics (237 citations), Pathology and Forensic Medicine (155 citations), Immunology (140 citations), Hematology (74 citations) and Radiology, Nuclear Medicine and Imaging (50 citations). Kerstin Willander has collaborated with scholars based in Sweden, United Kingdom and Finland. Frequent co-authors include Gerard Tobin, Peter Söderkvist, Richard Rosenquist, Ola Söderberg, Anita Söderberg, Sohvi Hörkkö, Eva Hellqvist, Charlotte Dahle, Anders Rosén and E. Bäckman. Their work appears in journals such as European Journal Of Haematology, Biomarker Research, Blood, Oncotarget and BMC Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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