Kate E. Gilling

585 citations
17 papers · 467 · h-index 11

Impact in

Papers in

    • Neuroscience and Neuropharmacology Research 13
    • Neurotransmitter Receptor Influence on Behavior 4
    • Neuroscience and Neural Engineering 2
    • Ion channel regulation and function 7
    • Receptor Mechanisms and Signaling 4

Kate E. Gilling

17 papers receiving 460 citations

Peers

Kate E. Gilling
Comparison fields: 5 of 68
  • Biological Psychiatry 40
  • Cellular and Molecular Neuroscience 239
  • Behavioral Neuroscience 29
  • Pharmacology 83
  • Psychiatry and Mental health 50
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Katarína Lichnerová Czechia
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Citations per field
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Citations per year

Countries citing papers authored by Kate E. Gilling

Since Specialization
Citations

This map shows the geographic impact of Kate E. Gilling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate E. Gilling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate E. Gilling more than expected).

Fields of papers citing papers by Kate E. Gilling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate E. Gilling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate E. Gilling. The network helps show where Kate E. Gilling may publish in the future.

Co-authors

The 25 scholars most cited alongside Kate E. Gilling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kate E. Gilling Line = papers co-authored together Kate E. Gilling links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 2003105
2 200963
3 200455
4 201749
5 200735
6 201631
7 201230
8 200724
9 202015
10 201515
11 200814
12 200710
13 20146
14 20086
15 20126
16 20142
17 20041

About Kate E. Gilling

Kate E. Gilling is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Cognitive Neuroscience, Psychiatry and Mental health and Organic Chemistry, having authored 17 papers that have together received 467 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (13 papers), Ion channel regulation and function (7 papers), Receptor Mechanisms and Signaling (4 papers), Neurotransmitter Receptor Influence on Behavior (4 papers), Neural dynamics and brain function (3 papers), Neuroscience and Neural Engineering (2 papers), Epilepsy research and treatment (2 papers) and Memory and Neural Mechanisms (2 papers). The work is most often cited by research in Biological Psychiatry (40 citations), Cellular and Molecular Neuroscience (239 citations), Behavioral Neuroscience (29 citations), Pharmacology (83 citations) and Psychiatry and Mental health (50 citations). Kate E. Gilling has collaborated with scholars based in Germany, United States and Latvia. Frequent co-authors include Chris G. Parsons, Claudia Jatzke, Rainer Rupprecht, Mirko Hechenberger, Gerhard Rammes, W. Zieglgänsberger, Gerald Gimpl, Gerhard Hapfelmeier, Uta Ferrari and B Eisensamer. Their work appears in journals such as Journal of Neural Transmission, Neuroscience, Molecular Psychiatry, Neuropharmacology and Antioxidants and Redox Signaling.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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